907	PGE 2 promotes intestinal tumor growth by altering the expression of tumor suppressing and DNA repair genes. PGE 2 promotes intestinal tumor growth by altering the expression of tumor suppressing and DNA repair genes. PGE 2 promotes intestinal tumor growth by altering the expression of tumor suppressing and DNA repair genes. PGE 2 promotes intestinal tumor growth by altering the expression of tumor suppressing and DNA repair genes. PGE 2 promotes intestinal tumor growth by altering the expression of tumor suppressing and DNA repair genes. PGE2 Intestinal tumors Tumor growth Tumor suppressor genes DNA repair genes Gene expression Oncogenesis Inflammation Colorectal cancer Prostaglandin E2 Genetic alterations Cell proliferation Apoptosis Tumor microenvironment Cancer progression
350	Discrimination between the initiator and elongation tRNAs depends on the translation initiation factor IF3. Discrimination between the initiator and elongation tRNAs depends on the translation initiation factor IF3. Discrimination between the initiator and elongation tRNAs depends on the translation initiation factor IF3. Discrimination between the initiator and elongation tRNAs depends on the translation initiation factor IF3. Discrimination between the initiator and elongation tRNAs depends on the translation initiation factor IF3. Discrimination Initiator tRNA Elongation tRNA Translation Initiation Factor IF3 Protein Synthesis Ribosome Start Codon mRNA Aminoacyl-tRNA Codon Recognition Ternary Complex Fidelity Translation Initiation
230	Carriers of the alcohol aldehyde dehydrogenase deficiency mutation drink less that non-carries. Carriers of the alcohol aldehyde dehydrogenase deficiency mutation drink less that non-carries. Carriers of the alcohol aldehyde dehydrogenase deficiency mutation drink less that non-carries. Carriers of the alcohol aldehyde dehydrogenase deficiency mutation drink less that non-carries. Carriers of the alcohol aldehyde dehydrogenase deficiency mutation drink less that non-carries. Carriers Alcohol aldehyde dehydrogenase deficiency Mutation Drink less Non-carriers Genetic predisposition Alcohol consumption Enzyme deficiency ALDH2 Genetic variation
593	Incidence of heart failure decreased by 10% in women since 1979. Incidence of heart failure decreased by 10% in women since 1979. Incidence of heart failure decreased by 10% in women since 1979. Incidence of heart failure decreased by 10% in women since 1979. Incidence of heart failure decreased by 10% in women since 1979. Incidence Heart failure Decreased Women Since
1216	The extracellular domain of TMEM27 is cleaved in human beta cells. The extracellular domain of TMEM27 is cleaved in human beta cells. The extracellular domain of TMEM27 is cleaved in human beta cells. The extracellular domain of TMEM27 is cleaved in human beta cells. The extracellular domain of TMEM27 is cleaved in human beta cells. Extracellular domain TMEM27 Cleaved Human Beta cells Protein processing Membrane protein Cellular biology Diabetes Pancreatic islets
1337	Ubiquitin ligase UBC13 generates a K63-linked polyubiquitin moiety at PCNA K164. Ubiquitin ligase UBC13 generates a K63-linked polyubiquitin moiety at PCNA K164. Ubiquitin ligase UBC13 generates a K63-linked polyubiquitin moiety at PCNA K164. Ubiquitin ligase UBC13 generates a K63-linked polyubiquitin moiety at PCNA K164. Ubiquitin ligase UBC13 generates a K63-linked polyubiquitin moiety at PCNA K164. Ubiquitin ligase UBC13 K63-linked Polyubiquitin PCNA K164 Ubiquitination Protein modification DNA repair Cell cycle regulation
232	Cataract and trachoma are the primary cause of blindness in Southern Sudan. Cataract and trachoma are the primary cause of blindness in Southern Sudan. Cataract and trachoma are the primary cause of blindness in Southern Sudan. Cataract and trachoma are the primary cause of blindness in Southern Sudan. Cataract and trachoma are the primary cause of blindness in Southern Sudan. Cataract Trachoma Blindness Southern Sudan Primary cause Eye diseases Vision impairment Public health Ophthalmology Preventive measures
1336	UCB T cells reduce TCR diversity after transplantation. UCB T cells reduce TCR diversity after transplantation. UCB T cells reduce TCR diversity after transplantation. UCB T cells reduce TCR diversity after transplantation. UCB T cells reduce TCR diversity after transplantation. UCB T cells TCR Diversity Transplantation Reduction Immune Chimerism Repertoire Graft Therapy Hematopoietic Stem cells Immunosuppression Lymphocytes Reconstitution Clinical Allogeneic Conditioning Outcome
233	Cell autonomous sex determination in somatic cells does not occur in Galliformes. Cell autonomous sex determination in somatic cells does not occur in Galliformes. Cell autonomous sex determination in somatic cells does not occur in Galliformes. Cell autonomous sex determination in somatic cells does not occur in Galliformes. Cell autonomous sex determination in somatic cells does not occur in Galliformes. Cell autonomous sex determination Somatic cells Galliformes Sex determination Non-cell autonomous Avian sex determination Somatic sex differentiation Bird sex development
354	Downregulation and mislocalization of Scribble prevents cell transformation and mammary tumorigenesis. Downregulation and mislocalization of Scribble prevents cell transformation and mammary tumorigenesis. Downregulation and mislocalization of Scribble prevents cell transformation and mammary tumorigenesis. Downregulation and mislocalization of Scribble prevents cell transformation and mammary tumorigenesis. Downregulation and mislocalization of Scribble prevents cell transformation and mammary tumorigenesis. Downregulation Mislocalization Scribble Cell transformation Mammary tumorigenesis Cancer Tumor suppression Protein localization Cellular signaling Epithelial cells Oncogenesis Cell polarity Molecular biology Tumor biology Breast cancer Genetic alterations Cell adhesion Tumor progression Biological pathways Cancer research
475	Glycolysis is one of the primary glycometabolic pathways in cells. Glycolysis is one of the primary glycometabolic pathways in cells. Glycolysis is one of the primary glycometabolic pathways in cells. Glycolysis is one of the primary glycometabolic pathways in cells. Glycolysis is one of the primary glycometabolic pathways in cells. Glycolysis Glycometabolic Pathways Cells Primary Metabolism Biochemistry Cellular Glucose Energy Anabolism Catabolism Enzymes ATP Pyruvate
113	Angiotensin converting enzyme inhibitors are associated with increased risk for functional renal insufficiency. Angiotensin converting enzyme inhibitors are associated with increased risk for functional renal insufficiency. Angiotensin converting enzyme inhibitors are associated with increased risk for functional renal insufficiency. Angiotensin converting enzyme inhibitors are associated with increased risk for functional renal insufficiency. Angiotensin converting enzyme inhibitors are associated with increased risk for functional renal insufficiency. Angiotensin Converting Enzyme Inhibitors ACE Risk Functional Renal Insufficiency Kidney Dysfunction Hypertension Cardiovascular Side Effects Medication Treatment Clinical Outcomes Pharmacology
1335	UCB T cells maintain high TCR diversity after transplantation. UCB T cells maintain high TCR diversity after transplantation. UCB T cells maintain high TCR diversity after transplantation. UCB T cells maintain high TCR diversity after transplantation. UCB T cells maintain high TCR diversity after transplantation. UCB T cells TCR Diversity Transplantation Maintenance Immunology Hematopoietic Stem cells Graft Chimerism Immune reconstitution Therapy Clinical Bioengineering Leakage Repertoire Lymphocytes Donor Recipient
597	Incidence rates of cervical cancer have decreased. Incidence rates of cervical cancer have decreased. Incidence rates of cervical cancer have decreased. Incidence rates of cervical cancer have decreased. Incidence rates of cervical cancer have decreased. incidence rates cervical cancer decreased reduction decline trends epidemiology public health prevention screening vaccination
1213	The deregulated and prolonged activation of monocytes has deleterious effects in inflammatory diseases. The deregulated and prolonged activation of monocytes has deleterious effects in inflammatory diseases. The deregulated and prolonged activation of monocytes has deleterious effects in inflammatory diseases. The deregulated and prolonged activation of monocytes has deleterious effects in inflammatory diseases. The deregulated and prolonged activation of monocytes has deleterious effects in inflammatory diseases. Deregulated Prolonged Activation Monocytes Deleterious Effects Inflammatory Diseases
598	Incidence rates of cervical cancer have increased due to nationwide screening programs based primarily on cytology to detect uterine cervical cancer. Incidence rates of cervical cancer have increased due to nationwide screening programs based primarily on cytology to detect uterine cervical cancer. Incidence rates of cervical cancer have increased due to nationwide screening programs based primarily on cytology to detect uterine cervical cancer. Incidence rates of cervical cancer have increased due to nationwide screening programs based primarily on cytology to detect uterine cervical cancer. Incidence rates of cervical cancer have increased due to nationwide screening programs based primarily on cytology to detect uterine cervical cancer. Cervical cancer Incidence rates Nationwide screening programs Cytology Detection Uterine cervical cancer Screening Cancer rates Cytological screening Public health programs
115	Anthrax spores can be disposed of easily after they are dispersed. Anthrax spores can be disposed of easily after they are dispersed. Anthrax spores can be disposed of easily after they are dispersed. Anthrax spores can be disposed of easily after they are dispersed. Anthrax spores can be disposed of easily after they are dispersed. Anthrax Spores Disposal Dispersal Containment Decontamination Biohazard Environmental Safety Public Health Biowaste Cleaning Treatment Sterilization Pathogens
236	Cell autonomous sex determination in somatic cells occurs in Passeriformes. Cell autonomous sex determination in somatic cells occurs in Passeriformes. Cell autonomous sex determination in somatic cells occurs in Passeriformes. Cell autonomous sex determination in somatic cells occurs in Passeriformes. Cell autonomous sex determination in somatic cells occurs in Passeriformes. Certainly! Here is a list of keywords for the given query Cell autonomous Sex determination Somatic cells Passeriformes Birds Genomic imprinting Sex chromosomes Developmental biology Genetic mechanisms Avian biology
478	Golli-deficient T-cells prefer to differentiate into an anergic phenotype in the adaptive immune response when there are increased levels of Ca2+ in the cytosol. Golli-deficient T-cells prefer to differentiate into an anergic phenotype in the adaptive immune response when there are increased levels of Ca2+ in the cytosol. Golli-deficient T-cells prefer to differentiate into an anergic phenotype in the adaptive immune response when there are increased levels of Ca2+ in the cytosol. Golli-deficient T-cells prefer to differentiate into an anergic phenotype in the adaptive immune response when there are increased levels of Ca2+ in the cytosol. Golli-deficient T-cells prefer to differentiate into an anergic phenotype in the adaptive immune response when there are increased levels of Ca2+ in the cytosol. Golli-deficient T-cells Differentiation Anergic phenotype Adaptive immune response Increased Ca2+ levels Cytosol concentration Immune tolerance Calcium signaling T-cell activation
1332	Tumor necrosis factor alpha (TNF-α) and interleukin-1 (IL-1) are pro-inflammatory cytokines that inhibit IL-6 and IL-10. Tumor necrosis factor alpha (TNF-α) and interleukin-1 (IL-1) are pro-inflammatory cytokines that inhibit IL-6 and IL-10. Tumor necrosis factor alpha (TNF-α) and interleukin-1 (IL-1) are pro-inflammatory cytokines that inhibit IL-6 and IL-10. Tumor necrosis factor alpha (TNF-α) and interleukin-1 (IL-1) are pro-inflammatory cytokines that inhibit IL-6 and IL-10. Tumor necrosis factor alpha (TNF-α) and interleukin-1 (IL-1) are pro-inflammatory cytokines that inhibit IL-6 and IL-10. Certainly! Here is a list of keywords for the given query Tumor Necrosis Factor Alpha TNF-α Interleukin-1 IL-1 Pro-inflammatory Cytokines Inhibit IL-6 IL-10 Cytokine Interaction Inflammation Regulation These keywords can help in searching for and organizing information related to the role of TNF-α and IL-1 in the regulation of other cytokines like IL-6 and IL-10
237	"Cells lacking clpC have a defect in sporulation efficiency in Bacillus subtilis. Cells lacking clpC have a defect in sporulation efficiency in Bacillus subtilis. Cells lacking clpC have a defect in sporulation efficiency in Bacillus subtilis. Cells lacking clpC have a defect in sporulation efficiency in Bacillus subtilis. Cells lacking clpC have a defect in sporulation efficiency in Bacillus subtilis. Here is a list of keywords for the query: ""Cells lastIndexing clpC have a defect in sporulation efficiency in Bacillus subtilis Cells clpC Defect Sporulation Efficiency Bacillus subtilis These keywords capture the essential elements of the query including the organism (Bacillus subtilis) the specific gene (clpC) and the observed biological phenomenon (defect in sporulation efficiency)"
238	Cells undergoing methionine restriction may activate miRNAs. Cells undergoing methionine restriction may activate miRNAs. Cells undergoing methionine restriction may activate miRNAs. Cells undergoing methionine restriction may activate miRNAs. Cells undergoing methionine restriction may activate miRNAs. Certainly! Here is a list of keywords for the given query Cells Methionine Restriction miRNAs Activation Gene Expression Metabolic Regulation Amino Acid Deprivation MicroRNA Cellular Response Epigenetic Regulation Nutrient Signaling Molecular Biology Biochemistry Metabolism These keywords should help in understanding and researching the topic of cells undergoing methionine restriction and the potential activation of miRNAs
118	Antibiotic induced alterations in the gut microbiome reduce resistance against Clostridium difficile Antibiotic induced alterations in the gut microbiome reduce resistance against Clostridium difficile Antibiotic induced alterations in the gut microbiome reduce resistance against Clostridium difficile Antibiotic induced alterations in the gut microbiome reduce resistance against Clostridium difficile Antibiotic induced alterations in the gut microbiome reduce resistance against Clostridium difficile Antibiotic Gut microbiome Alterations Resistance Clostridium difficile Microbial ecology Intestinal flora Pathogenesis Dysbiosis Probiotics Bacterial infection Gastrointestinal tract Symbiosis Antibiotic-associated diarrhea Microbiota Health Disease Microbial diversity Therapeutics Prevention
239	Cellular aging closely links to an older appearance. Cellular aging closely links to an older appearance. Cellular aging closely links to an older appearance. Cellular aging closely links to an older appearance. Cellular aging closely links to an older appearance. Cellular aging Older appearance Skin aging Telomere shortening Senescent cells Collagen degradation Elastin fibers Facial wrinkles Age spots Cellular metabolism DNA damage Free radicals Antioxidants Hormonal changes Epidermal thinning Dermal atrophy Aging biomarkers Oxidative stress Genetic factors Environmental influences
911	PKG-la plays an essential role in expression of pain hypersensitivity in PGK-la knockout mice. PKG-la plays an essential role in expression of pain hypersensitivity in PGK-la knockout mice. PKG-la plays an essential role in expression of pain hypersensitivity in PGK-la knockout mice. PKG-la plays an essential role in expression of pain hypersensitivity in PGK-la knockout mice. PKG-la plays an essential role in expression of pain hypersensitivity in PGK-la knockout mice. PKG-la Pain hypersensitivity Expression Knockout mice Role Essential PGK-la
913	PPAR-RXRs are inhibited by PPAR ligands. PPAR-RXRs are inhibited by PPAR ligands. PPAR-RXRs are inhibited by PPAR ligands. PPAR-RXRs are inhibited by PPAR ligands. PPAR-RXRs are inhibited by PPAR ligands. PPAR RXRs Inhibited PPAR ligands Ligands Inhibition Interaction Receptors Molecular biology Pharmacology
914	PPAR-RXRs can be activated by PPAR ligands. PPAR-RXRs can be activated by PPAR ligands. PPAR-RXRs can be activated by PPAR ligands. PPAR-RXRs can be activated by PPAR ligands. PPAR-RXRs can be activated by PPAR ligands. PPAR RXRs Activation PPAR ligands Molecular biology Gene expression Nuclear receptors Drug targets Metabolic diseases Hormone signaling
1339	Ultrasound guidance significantly raises the number of traumatic procedures when attempting needle insertion. Ultrasound guidance significantly raises the number of traumatic procedures when attempting needle insertion. Ultrasound guidance significantly raises the number of traumatic procedures when attempting needle insertion. Ultrasound guidance significantly raises the number of traumatic procedures when attempting needle insertion. Ultrasound guidance significantly raises the number of traumatic procedures when attempting needle insertion. Ultrasound guidance Traumatic procedures Needle insertion Medical procedures Patient safety Clinical outcomes Interventions Imaging technology Healthcare quality Procedural complications
13	"5% of perinatal mortality is due to low birth weight. 5% of perinatal mortality is due to low birth weight. 5% of perinatal mortality is due to low birth weight. 5% of perinatal mortality is due to low birth weight. 5% of perinatal mortality is due to low birth weight. Certainly! Here is a list of keywords for the query ""5% of perinatal mortality is due to low birth weight"" Perinatal Mortality Low birth weight Percentage Neonatal Infant health Obstetrics Prenatal care Maternal health Epidemiology These keywords can help in conducting further research or organizing information related to the topic"
1110	Suboptimal nutrition is not predictive of chronic disease Suboptimal nutrition is not predictive of chronic disease Suboptimal nutrition is not predictive of chronic disease Suboptimal nutrition is not predictive of chronic disease Suboptimal nutrition is not predictive of chronic disease Suboptimal nutrition Chronic disease Predictive Nutritional status Health outcomes Dietary intake Long-term health Risk factors Epidemiology Nutritional epidemiology Public health Preventive medicine Biomarkers Nutrient deficiencies Malnutrition Chronic conditions Health indicators Epidemiological studies Nutritional assessment Disease prevention
1352	Upregulation of mosGCTL-1 is induced upon infection with West Nile virus. Upregulation of mosGCTL-1 is induced upon infection with West Nile virus. Upregulation of mosGCTL-1 is induced upon infection with West Nile virus. Upregulation of mosGCTL-1 is induced upon infection with West Nile virus. Upregulation of mosGCTL-1 is induced upon infection with West Nile virus. Upregulation mosGCTL-1 Induced Infection West Nile virus Viral infection Gene expression Host response Immune response Pathogen interaction Viral pathogenesis Molecular mechanism Cell signaling Protein expression Viral genetics Host genetics RNA expression Gene regulation Pathogen-induced genes Antiviral response
362	During the primary early antibody response activated B cells migrate toward the inner-and outer paracortical areas where oxysterol accumulation is generated by stromal cells. During the primary early antibody response activated B cells migrate toward the inner-and outer paracortical areas where oxysterol accumulation is generated by stromal cells. During the primary early antibody response activated B cells migrate toward the inner-and outer paracortical areas where oxysterol accumulation is generated by stromal cells. During the primary early antibody response activated B cells migrate toward the inner-and outer paracortical areas where oxysterol accumulation is generated by stromal cells. During the primary early antibody response activated B cells migrate toward the inner-and outer paracortical areas where oxysterol accumulation is generated by stromal cells. Primary Early Antibody Response Activated B cells Migrate Inner paracortical Outer paracortical Areas Oxysterol Accumulation Stromal cells
1107	Subcutaneous fat depots undergo extensive browning processes after cold exposure. Subcutaneous fat depots undergo extensive browning processes after cold exposure. Subcutaneous fat depots undergo extensive browning processes after cold exposure. Subcutaneous fat depots undergo extensive browning processes after cold exposure. Subcutaneous fat depots undergo extensive browning processes after cold exposure. Subcutaneous fat Fat depots Browning Cold exposure Thermogenesis Adipose tissue White adipose tissue Brown adipose tissue Metabolism Energy expenditure Cold-induced browning Temperature regulation Adipocytes Beige fat
1	0-dimensional biomaterials show inductive properties. 0-dimensional biomaterials show inductive properties. 0-dimensional biomaterials show inductive properties. 0-dimensional biomaterials show inductive properties. 0-dimensional biomaterials show inductive properties. dimensional biomaterials inductive properties nanoparticles biocompatibility cellular interaction tissue engineering drug delivery
1226	The loss of the TET protein functions may have dire biological consequences, such as myeloid cancers. The loss of the TET protein functions may have dire biological consequences, such as myeloid cancers. The loss of the TET protein functions may have dire biological consequences, such as myeloid cancers. The loss of the TET protein functions may have dire biological consequences, such as myeloid cancers. The loss of the TET protein functions may have dire biological consequences, such as myeloid cancers. TET protein Loss of function Biological consequences Myeloid cancers Protein dysfunction Cancer development Genetic disorders Epigenetic regulation DNA modification Hematopoietic system
1104	Stroke patients with prior use of direct oral anticoagulants have a lower risk of in-hospital mortality than stroke patients with prior use of warfarin. Stroke patients with prior use of direct oral anticoagulants have a lower risk of in-hospital mortality than stroke patients with prior use of warfarin. Stroke patients with prior use of direct oral anticoagulants have a lower risk of in-hospital mortality than stroke patients with prior use of warfarin. Stroke patients with prior use of direct oral anticoagulants have a lower risk of in-hospital mortality than stroke patients with prior use of warfarin. Stroke patients with prior use of direct oral anticoagulants have a lower risk of in-hospital mortality than stroke patients with prior use of warfarin. Stroke Direct Oral Anticoagulants Warfarin In-hospital Mortality Prior Use Anticoagulation Risk Reduction Thromboembolic Disease Stroke Prognosis Comparative Effectiveness Hospital Outcomes Anticoagulant Therapy Cardiovascular Patients Clinical Outcomes Medication History
1225	The locus rs647161 is associated with colorectal carcinoma. The locus rs647161 is associated with colorectal carcinoma. The locus rs647161 is associated with colorectal carcinoma. The locus rs647161 is associated with colorectal carcinoma. The locus rs647161 is associated with colorectal carcinoma. Locus rs647161 Colorectal Carcinoma Association Genetic SNP Risk Cancer Polymorphism
124	Antiretroviral therapy reduces rates of tuberculosis across a broad range of CD4 strata. Antiretroviral therapy reduces rates of tuberculosis across a broad range of CD4 strata. Antiretroviral therapy reduces rates of tuberculosis across a broad range of CD4 strata. Antiretroviral therapy reduces rates of tuberculosis across a broad range of CD4 strata. Antiretroviral therapy reduces rates of tuberculosis across a broad range of CD4 strata. Antiretroviral therapy Tuberculosis CD4 strata Rates reduction HIV Immune system T-cell count Treatment efficacy Public health Clinical outcomes
3	1,000 genomes project enables mapping of genetic sequence variation consisting of rare variants with larger penetrance effects than common variants. 1,000 genomes project enables mapping of genetic sequence variation consisting of rare variants with larger penetrance effects than common variants. 1,000 genomes project enables mapping of genetic sequence variation consisting of rare variants with larger penetrance effects than common variants. 1,000 genomes project enables mapping of genetic sequence variation consisting of rare variants with larger penetrance effects than common variants. 1,000 genomes project enables mapping of genetic sequence variation consisting of rare variants with larger penetrance effects than common variants. 000 Genomes Project Genetic Sequence Variation Rare Variants Common Variants Penetrance Effects Genomic Mapping Genetic Diversity Population Genetics Rare Alleles Functional Variants High-Impact Variants Genetic Disorders Genome-Wide Association Studies (GWAS) Personalized Medicine Genetic Epidemiology
1344	Up-regulation of the p53 pathway and related molecular events casues cancer resistance and results in a significantly shortened lifespan marked by senescent cells and accelerated organismal aging. Up-regulation of the p53 pathway and related molecular events casues cancer resistance and results in a significantly shortened lifespan marked by senescent cells and accelerated organismal aging. Up-regulation of the p53 pathway and related molecular events casues cancer resistance and results in a significantly shortened lifespan marked by senescent cells and accelerated organismal aging. Up-regulation of the p53 pathway and related molecular events casues cancer resistance and results in a significantly shortened lifespan marked by senescent cells and accelerated organismal aging. Up-regulation of the p53 pathway and related molecular events casues cancer resistance and results in a significantly shortened lifespan marked by senescent cells and accelerated organismal aging. Up-regulation p53 pathway Cancer resistance Molecular events Shortened lifespan Senescent cells Accelerated aging Organismal aging Cellular senescence p53 activation Aging mechanisms Tumor suppression Cellular pathways Biological aging Senescence-associated Lifespan reduction Molecular biology Age-related changes Cellular stress Gene
5	1/2000 in UK have abnormal PrP positivity. 1/2000 in UK have abnormal PrP positivity. 1/2000 in UK have abnormal PrP positivity. 1/2000 in UK have abnormal PrP positivity. 1/2000 in UK have abnormal PrP positivity. Abnormal PrP Positivity UK Prevalence Population 1/2000 Prion Disease Epidemiology Prion Protein Neurodegenerative Health Statistics Medical Research Public Health
127	Arginine 90 in p150n is important for interaction with EB1. Arginine 90 in p150n is important for interaction with EB1. Arginine 90 in p150n is important for interaction with EB1. Arginine 90 in p150n is important for interaction with EB1. Arginine 90 in p150n is important for interaction with EB1. Arginine p150n EB1 Interaction Protein Amino Acid Structural Biology Molecular Biology Cell Biology Neuroscience Microtubule Plus-End Tracking Protein Adaptor Protein Signaling Pathway Cellular Function Mutation Binding Site Residue Domain Protein-Protein Interaction Biochemistry
248	Chenodeosycholic acid treatment increases whole-body energy expenditure. Chenodeosycholic acid treatment increases whole-body energy expenditure. Chenodeosycholic acid treatment increases whole-body energy expenditure. Chenodeosycholic acid treatment increases whole-body energy expenditure. Chenodeosycholic acid treatment increases whole-body energy expenditure. Chenodeoxycholic acid Treatment Whole-body energy expenditure Metabolism Thermogenesis Hormonal regulation Bile acids Energy metabolism Biochemical pathways Physiological response Clinical trials Obesity Weight management Fat oxidation Nutrient metabolism
1100	Statins increase blood cholesterol. Statins increase blood cholesterol. Statins increase blood cholesterol. Statins increase blood cholesterol. Statins increase blood cholesterol. Statins Blood Cholesterol Increase Effect Lipid Cardiovascular Drug Treatment Mechanism
1221	The genomic aberrations found in matasteses are very similar to those found in the primary tumor. The genomic aberrations found in matasteses are very similar to those found in the primary tumor. The genomic aberrations found in matasteses are very similar to those found in the primary tumor. The genomic aberrations found in matasteses are very similar to those found in the primary tumor. The genomic aberrations found in matasteses are very similar to those found in the primary tumor. Genomic Aberrations Metastases Primary Tumor Similar Genetic Mutations Cancer Tissue DNA Prognosis Treatment Biomarkers Comparative Oncology Genotype Phenotype Tumor Evolution Molecular Profiling
128	Arterioles have a larger lumen diameter than venules. Arterioles have a larger lumen diameter than venules. Arterioles have a larger lumen diameter than venules. Arterioles have a larger lumen diameter than venules. Arterioles have a larger lumen diameter than venules. Arterioles Venules Lumen Diameter Blood vessels Circulatory system Comparison Size Anatomy Physiology
249	Chenodeosycholic acid treatment reduces whole-body energy expenditure. Chenodeosycholic acid treatment reduces whole-body energy expenditure. Chenodeosycholic acid treatment reduces whole-body energy expenditure. Chenodeosycholic acid treatment reduces whole-body energy expenditure. Chenodeosycholic acid treatment reduces whole-body energy expenditure. Chenodeoxycholic acid Treatment Whole-body energy expenditure Metabolism Bile acids Caloric output Thermogenesis Energy metabolism Reduction Physiological response
129	Articles published in open access format are less likely to be cited than traditional journals. Articles published in open access format are less likely to be cited than traditional journals. Articles published in open access format are less likely to be cited than traditional journals. Articles published in open access format are less likely to be cited than traditional journals. Articles published in open access format are less likely to be cited than traditional journals. Open access Traditional journals Citation impact Academic publishing Research dissemination Peer-reviewed articles Article influence Scholarly communication Open access journals Citation frequency Publication bias Research visibility Access barriers Scientific impact Citation analysis
800	Modifying the epigenome in the brain affects the normal human aging process by affecting certain genes related to neurogenesis. Modifying the epigenome in the brain affects the normal human aging process by affecting certain genes related to neurogenesis. Modifying the epigenome in the brain affects the normal human aging process by affecting certain genes related to neurogenesis. Modifying the epigenome in the brain affects the normal human aging process by affecting certain genes related to neurogenesis. Modifying the epigenome in the brain affects the normal human aging process by affecting certain genes related to neurogenesis. Epigenome Brain Human aging Genes Neurogenesis Epigenetic modification Neural stem cells Gene expression Aging process Brain function Epigenetic marks DNA methylation Histone modification Neural plasticity Cognitive decline Cellular senescence Neurodegeneration Epigenetic regulators Lifespan Brain health
921	Participating in six months of physical activity improves cognitive functioning. Participating in six months of physical activity improves cognitive functioning. Participating in six months of physical activity improves cognitive functioning. Participating in six months of physical activity improves cognitive functioning. Participating in six months of physical activity improves cognitive functioning. Physical activity Cognitive functioning Improvement Six months Exercise Mental health Brain function Long-term effects Fitness Cognitive enhancement
922	Patients in stable partnerships have a faster progression from HIV to AIDS. Patients in stable partnerships have a faster progression from HIV to AIDS. Patients in stable partnerships have a faster progression from HIV to AIDS. Patients in stable partnerships have a faster progression from HIV to AIDS. Patients in stable partnerships have a faster progression from HIV to AIDS. Patients Stable partnerships Faster progression HIV AIDS Relationship status Disease progression Viral infection Partners Health outcomes Epidemiology Sexual behavior Support systems Long-term relationships Immunological response
805	Monoclonal antibody targeting of N-cadherin inhibits metastasis. Monoclonal antibody targeting of N-cadherin inhibits metastasis. Monoclonal antibody targeting of N-cadherin inhibits metastasis. Monoclonal antibody targeting of N-cadherin inhibits metastasis. Monoclonal antibody targeting of N-cadherin inhibits metastasis. Monoclonal antibody N-cadherin Metastasis inhibition Targeted therapy Cancer metastasis N-cadherin targeting Antibody therapy Cancer treatment Metastatic inhibition Cadherin-targeting antibodies
808	Most termination events in Okazaki fragments are sequence specific. Most termination events in Okazaki fragments are sequence specific. Most termination events in Okazaki fragments are sequence specific. Most termination events in Okazaki fragments are sequence specific. Most termination events in Okazaki fragments are sequence specific. Okazaki fragments Termination events Sequence specific DNA replication Lagging strand Nucleotide sequences Enzymes DNA synthesis Molecular biology
1121	Synaptic activity enhances local release of brain derived neurotrophic factor from postsynaptic dendrites. Synaptic activity enhances local release of brain derived neurotrophic factor from postsynaptic dendrites. Synaptic activity enhances local release of brain derived neurotrophic factor from postsynaptic dendrites. Synaptic activity enhances local release of brain derived neurotrophic factor from postsynaptic dendrites. Synaptic activity enhances local release of brain derived neurotrophic factor from postsynaptic dendrites. Synaptic activity Brain-derived neurotrophic factor (BDNF) Postsynaptic dendrites Local release Neuroplasticity Synaptic transmission Dendritic signaling Neurotrophic signaling Synaptic strength Dendritic spines BDNF release Postsynaptic mechanisms Neurotrophic factors Synaptic enhancement Local BDNF release
1363	Venules have a thinner or absent smooth layer compared to arterioles. Venules have a thinner or absent smooth layer compared to arterioles. Venules have a thinner or absent smooth layer compared to arterioles. Venules have a thinner or absent smooth layer compared to arterioles. Venules have a thinner or absent smooth layer compared to arterioles. Venules Thinner Absent Smooth Muscle Layer Arterioles Comparison Vascular Structure Blood Vessels Circulatory System Anatomy Histology Physiology Blood Flow Vascular Wall Tissue Structure
1241	The myocardial lineage develops from cardiac progenitors of mesodermal origin. The myocardial lineage develops from cardiac progenitors of mesodermal origin. The myocardial lineage develops from cardiac progenitors of mesodermal origin. The myocardial lineage develops from cardiac progenitors of mesodermal origin. The myocardial lineage develops from cardiac progenitors of mesodermal origin. Myocardial lineage Cardiac progenitors Mesodermal origin Heart development Cardiac mesoderm Embryonic development Cardiogenesis Myocardium Progenitor cells Mesoderm differentiation
1362	Venules have a larger lumen diameter than arterioles. Venules have a larger lumen diameter than arterioles. Venules have a larger lumen diameter than arterioles. Venules have a larger lumen diameter than arterioles. Venules have a larger lumen diameter than arterioles. Venules Larger Lumen Diameter Arterioles
491	HNF4A mutations can cause diabetes in mutant carriers by the age of 14 years HNF4A mutations can cause diabetes in mutant carriers by the age of 14 years HNF4A mutations can cause diabetes in mutant carriers by the age of 14 years HNF4A mutations can cause diabetes in mutant carriers by the age of 14 years HNF4A mutations can cause diabetes in mutant carriers by the age of 14 years HNF4A Mutations Diabetes Mutant carriers Age 14 years Genetic disorder Early-onset diabetes Hepatocyte nuclear factor 4 alpha Molecular genetics
130	Articles published in open access format are more likely to be cited than traditional journals. Articles published in open access format are more likely to be cited than traditional journals. Articles published in open access format are more likely to be cited than traditional journals. Articles published in open access format are more likely to be cited than traditional journals. Articles published in open access format are more likely to be cited than traditional journals. Certainly! Here is a list of keywords for the given query Open Access Traditional Journals Citation Impact Academic Publishing Research Dissemination Scholarly Communication Publication Models Visibility Accessibility Peer Review Scientific Impact Open Science Citation Metrics Publication Bias Reader Engagement These keywords can help in conducting a comprehensive search or analysis related to the impact of open access publication on citation rates
132	"Aspirin inhibits the production of PGE2. Aspirin inhibits the production of PGE2. Aspirin inhibits the production of PGE2. Aspirin inhibits the production of PGE2. Aspirin inhibits the production of PGE2. Certainly! Here is a list of keywords for the query ""Aspirin inhibits the production of PGE2"" Aspirin Inhibits Production PGE2 Prostaglandin E2 Anti-inflammatory COX Cyclooxygenase NSAID Nonsteroidal anti-inflammatory drug These keywords can help in searching for more detailed information or conducting further research on the topic"
133	Assembly of invadopodia is triggered by focal generation of phosphatidylinositol-3,4-biphosphate and the activation of the nonreceptor tyrosine kinase Src. Assembly of invadopodia is triggered by focal generation of phosphatidylinositol-3,4-biphosphate and the activation of the nonreceptor tyrosine kinase Src. Assembly of invadopodia is triggered by focal generation of phosphatidylinositol-3,4-biphosphate and the activation of the nonreceptor tyrosine kinase Src. Assembly of invadopodia is triggered by focal generation of phosphatidylinositol-3,4-biphosphate and the activation of the nonreceptor tyrosine kinase Src. Assembly of invadopodia is triggered by focal generation of phosphatidylinositol-3,4-biphosphate and the activation of the nonreceptor tyrosine kinase Src. Invadopodia Assembly Phosphatidylinositol-3 4-bisphosphate Focal generation Nonreceptor tyrosine kinase Src Activation Signaling Cell invasion Cancer metastasis
1359	Varenicline monotherapy is more effective after 12 weeks of treatment compared to combination nicotine replacement therapies with varenicline or bupropion. Varenicline monotherapy is more effective after 12 weeks of treatment compared to combination nicotine replacement therapies with varenicline or bupropion. Varenicline monotherapy is more effective after 12 weeks of treatment compared to combination nicotine replacement therapies with varenicline or bupropion. Varenicline monotherapy is more effective after 12 weeks of treatment compared to combination nicotine replacement therapies with varenicline or bupropion. Varenicline monotherapy is more effective after 12 weeks of treatment compared to combination nicotine replacement therapies with varenicline or bupropion. Certainly! Here is a list of keywords based on the provided query Varenicline Monotherapy Effectiveness 12 weeks Treatment Combination therapy Nicotine replacement therapy Bupropion Smoking cessation Clinical outcomes Comparative study Pharmacotherapy Smoking cessation aids Therapeutic response Drug efficacy
137	Asymptomatic visual impairment screening in elderly populations does not lead to improved vision. Asymptomatic visual impairment screening in elderly populations does not lead to improved vision. Asymptomatic visual impairment screening in elderly populations does not lead to improved vision. Asymptomatic visual impairment screening in elderly populations does not lead to improved vision. Asymptomatic visual impairment screening in elderly populations does not lead to improved vision. Certainly! Here is a list of keywords for the given query Asymptomatic Visual impairment Screening Elderly Populations Improved vision Eye health Geriatric Optometry Ophthalmology Vision testing Preventive care Health outcomes Medical screening Age-related visual issues
1232	The minor G allele of FOXO3 is related to more severe symptoms of Crohn's Disease. The minor G allele of FOXO3 is related to more severe symptoms of Crohn's Disease. The minor G allele of FOXO3 is related to more severe symptoms of Crohn's Disease. The minor G allele of FOXO3 is related to more severe symptoms of Crohn's Disease. The minor G allele of FOXO3 is related to more severe symptoms of Crohn's Disease. Certainly! Here is a list of keywords for the given query Minor G allele FOXO3 gene Crohn's Disease Genetic association Disease severity Symptom intensity Genetic polymorphism Inflammatory bowel disease (IBD) Genotype-phenotype correlation Genetic marker
811	Mutant mice lacking SVCT2 have greatly increased ascorbic acid levels in both brain and adrenals. Mutant mice lacking SVCT2 have greatly increased ascorbic acid levels in both brain and adrenals. Mutant mice lacking SVCT2 have greatly increased ascorbic acid levels in both brain and adrenals. Mutant mice lacking SVCT2 have greatly increased ascorbic acid levels in both brain and adrenals. Mutant mice lacking SVCT2 have greatly increased ascorbic acid levels in both brain and adrenals. Mutant mice SVCT2 Lacking SVCT2 Ascorbic acid Brain Adrenals Increased ascorbic acid levels Vitamin C SVCT2 deficiency Mouse model Neurological impact Endocrine impact Ascorbic acid metabolism SVCT2 knockout Tissue-specific ascorbic acid Biological mechanisms Genetic modification Ascorbic acid transport
814	Mutations in G-Beta protein GNB2 are present in many cancers, resulting in loss of interaction with G-alpha subunits and concomitant activation of AKT pathway. Mutations in G-Beta protein GNB2 are present in many cancers, resulting in loss of interaction with G-alpha subunits and concomitant activation of AKT pathway. Mutations in G-Beta protein GNB2 are present in many cancers, resulting in loss of interaction with G-alpha subunits and concomitant activation of AKT pathway. Mutations in G-Beta protein GNB2 are present in many cancers, resulting in loss of interaction with G-alpha subunits and concomitant activation of AKT pathway. Mutations in G-Beta protein GNB2 are present in many cancers, resulting in loss of interaction with G-alpha subunits and concomitant activation of AKT pathway. Certainly! Here are the keywords for the given query Mutations G-Beta protein GNB2 Cancers Loss of interaction G-alpha subunits AKT pathway Concomitant activation
936	"Peroxynitrite is required for nitration of TCR/CD8. Peroxynitrite is required for nitration of TCR/CD8. Peroxynitrite is required for nitration of TCR/CD8. Peroxynitrite is required for nitration of TCR/CD8. Peroxynitrite is required for nitration of TCR/CD8. Certainly! Here is a list of keywords for the query ""Peroxynitrite is required for nitration of TCR/CD8"" Peroxynitrite Nitration TCR CD8 T-cell receptor Cytotoxic T lymphocyte Reactive nitrogen species Post-translational modification Immune response Signal transduction These keywords can help in further researching the topic or understanding the context of the query"
36	A deficiency of vitamin B12 increases blood levels of homocysteine. A deficiency of vitamin B12 increases blood levels of homocysteine. A deficiency of vitamin B12 increases blood levels of homocysteine. A deficiency of vitamin B12 increases blood levels of homocysteine. A deficiency of vitamin B12 increases blood levels of homocysteine. Vitamin B12 Deficiency Blood levels Homocysteine Increase Nutritional deficiency Biochemistry Health impact Metabolic disorder Dietary intake
1132	TCR/CD3 microdomains are a required to induce the immunologic synapse to activate T cells. TCR/CD3 microdomains are a required to induce the immunologic synapse to activate T cells. TCR/CD3 microdomains are a required to induce the immunologic synapse to activate T cells. TCR/CD3 microdomains are a required to induce the immunologic synapse to activate T cells. TCR/CD3 microdomains are a required to induce the immunologic synapse to activate T cells. Certainly! Here is a list of keywords for the given query TCR CD3 Microdomains Immunologic Synapse T Cell Activation Required Induction T-Cell Receptor Cluster of Differentiation 3 Signaling Complex Immune Response Lymphocytes Cell Signaling Molecular Interactions Immunology
1130	T regulatory cells (tTregs) lacking αvβ8 are more adept at suppressing pathogenic T-cell responses during active inflammation. T regulatory cells (tTregs) lacking αvβ8 are more adept at suppressing pathogenic T-cell responses during active inflammation. T regulatory cells (tTregs) lacking αvβ8 are more adept at suppressing pathogenic T-cell responses during active inflammation. T regulatory cells (tTregs) lacking αvβ8 are more adept at suppressing pathogenic T-cell responses during active inflammation. T regulatory cells (tTregs) lacking αvβ8 are more adept at suppressing pathogenic T-cell responses during active inflammation. T regulatory cells tTregs αvβ8 Suppressing Pathogenic T-cell responses Active inflammation Immune response Immunology Cell signaling T-cell activation Inflammation regulation Lymphocytes Autoimmunity Cytokines Integrins Cell-mediated immunity T-cell subsets Pathogenesis Therapeutic targets Immune
380	Enhanced early production of inflammatory chemokines improves viral control in the lung. Enhanced early production of inflammatory chemokines improves viral control in the lung. Enhanced early production of inflammatory chemokines improves viral control in the lung. Enhanced early production of inflammatory chemokines improves viral control in the lung. Enhanced early production of inflammatory chemokines improves viral control in the lung. Inflammatory chemokines Early production Viral control Lung Enhanced response Immune response Antiviral defense Chemokine expression Respiratory infections Pathogen clearance
1370	Vitamin D deficiency is unrelated to birth weight. Vitamin D deficiency is unrelated to birth weight. Vitamin D deficiency is unrelated to birth weight. Vitamin D deficiency is unrelated to birth weight. Vitamin D deficiency is unrelated to birth weight. Vitamin D Deficiency Birth weight Unrelated Pregnancy Neonatal Nutritional status Maternal health Infants Correlation Clinical studies Epidemiology Health outcomes Pediatric Nutrient levels
261	Chronic aerobic exercise alters endothelial function, improving vasodilating mechanisms mediated by NO. Chronic aerobic exercise alters endothelial function, improving vasodilating mechanisms mediated by NO. Chronic aerobic exercise alters endothelial function, improving vasodilating mechanisms mediated by NO. Chronic aerobic exercise alters endothelial function, improving vasodilating mechanisms mediated by NO. Chronic aerobic exercise alters endothelial function, improving vasodilating mechanisms mediated by NO. Chronic Aerobic Exercise Endothelial Function Vasodilation Mechanisms Nitric Oxide NO Improvement Cardiovascular Health Blood Flow Vessels Physiology Molecular Signaling Pathways
141	Auditory entrainment is strengthened when people see congruent visual and auditory information. Auditory entrainment is strengthened when people see congruent visual and auditory information. Auditory entrainment is strengthened when people see congruent visual and auditory information. Auditory entrainment is strengthened when people see congruent visual and auditory information. Auditory entrainment is strengthened when people see congruent visual and auditory information. Auditory entrainment Visual information Auditory information Congruent stimuli Sensory integration Multisensory processing Visual-auditory interaction Synchronization Perceptual alignment Cross-modal effects
142	Autologous transplantation of mesenchymal stem cells causes a higher rate of opportunistic infections than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation of mesenchymal stem cells causes a higher rate of opportunistic infections than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation of mesenchymal stem cells causes a higher rate of opportunistic infections than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation of mesenchymal stem cells causes a higher rate of opportunistic infections than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation of mesenchymal stem cells causes a higher rate of opportunistic infections than induction therapy with anti-interleukin-2 receptor antibodies. Certainly! Here is a list of keywords for the given query Autologous transplantation Mesenchymal stem cells Opportunistic infections Induction therapy Anti-interleukin-2 receptor antibodies Higher rate Immune response Comparative study Stem cell therapy Immunotherapy
384	Epidemiological disease burden from noncommunicable diseases is more prevalent in low economic settings. Epidemiological disease burden from noncommunicable diseases is more prevalent in low economic settings. Epidemiological disease burden from noncommunicable diseases is more prevalent in low economic settings. Epidemiological disease burden from noncommunicable diseases is more prevalent in low economic settings. Epidemiological disease burden from noncommunicable diseases is more prevalent in low economic settings. Certainly! Here is a list of keywords for the query Epidemiological Disease burden Noncommunicable diseases Low economic settings Prevalence Health disparities Socioeconomic status Public health Chronic diseases Global health Poverty Risk factors Health outcomes Health inequality Low-income countries NCDs (Non-Communicable Diseases) Prevention Healthcare access Economic impact
143	Autologous transplantation of mesenchymal stem cells causes fewer opportunistic infections than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation of mesenchymal stem cells causes fewer opportunistic infections than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation of mesenchymal stem cells causes fewer opportunistic infections than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation of mesenchymal stem cells causes fewer opportunistic infections than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation of mesenchymal stem cells causes fewer opportunistic infections than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation Mesenchymal stem cells Opportunistic infections Induction therapy Anti-interleukin-2 receptor antibodies Comparative study Immunosuppression Stem cell therapy Clinical outcomes Infection risk Immune response Therapeutic approaches Hematopoietic stem cells Graft-versus-host disease Treatment efficacy
385	Epigenetic modulating agents (EMAs) modulate antitumor immune response in a cancer model system. Epigenetic modulating agents (EMAs) modulate antitumor immune response in a cancer model system. Epigenetic modulating agents (EMAs) modulate antitumor immune response in a cancer model system. Epigenetic modulating agents (EMAs) modulate antitumor immune response in a cancer model system. Epigenetic modulating agents (EMAs) modulate antitumor immune response in a cancer model system. Epigenetic modulating agents EMAs Antitumor immune response Cancer model system Epigenetic regulation Immune modulation Cancer immunotherapy Tumor microenvironment Epigenetics Immune cells Cancer treatment Gene expression Histone modification DNA methylation Chromatin structure Immune checkpoint inhibitors Cytotoxic T cells Dendritic cells
386	Errors in peripheral IV drug administration are most common during bolus administration and multiple-step medicine preparations. Errors in peripheral IV drug administration are most common during bolus administration and multiple-step medicine preparations. Errors in peripheral IV drug administration are most common during bolus administration and multiple-step medicine preparations. Errors in peripheral IV drug administration are most common during bolus administration and multiple-step medicine preparations. Errors in peripheral IV drug administration are most common during bolus administration and multiple-step medicine preparations. Errors Peripheral IV Drug administration Bolus administration Multiple-step Medicine preparations Common Intravenous Medication Infusion Clinical errors IV therapy Pharmaceutical Dosage Patient safety
1368	"Vitamin D deficiency effects the term of delivery. Vitamin D deficiency effects the term of delivery. Vitamin D deficiency effects the term of delivery. Vitamin D deficiency effects the term of delivery. Vitamin D deficiency effects the term of delivery. Certainly! Here is a list of keywords for the query ""Vitamin D deficiency effects the term of delivery"" Vitamin D Deficiency Pregnancy Term Delivery Preterm birth Gestation Maternal health Fetal development Nutritional status Obstetrics Complications Low birth weight Preeclampsia Neonatal outcomes Postpartum Hormonal imbalances Imm"
146	Autologous transplantation of mesenchymal stem cells has lower rates of rejection than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation of mesenchymal stem cells has lower rates of rejection than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation of mesenchymal stem cells has lower rates of rejection than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation of mesenchymal stem cells has lower rates of rejection than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation of mesenchymal stem cells has lower rates of rejection than induction therapy with anti-interleukin-2 receptor antibodies. Autologous transplantation Mesenchymal stem cells Rejection rates Induction therapy Anti-interleukin-2 receptor antibodies Comparative effectiveness Immune response Stem cell therapy Immunomodulation Clinical outcomes Transplant rejection Biological therapy Medical treatment Immunosuppression Therapeutic protocols
388	"Ethanol stress decreases the expression of IBP in bacteria. Ethanol stress decreases the expression of IBP in bacteria. Ethanol stress decreases the expression of IBP in bacteria. Ethanol stress decreases the expression of IBP in bacteria. Ethanol stress decreases the expression of IBP in bacteria. Certainly! Here is a list of keywords for the query ""Ethanol stress decreases the expression of IBP in bacteria"" Ethanol Stress IBP (Intrinsic Bacterial Proteins) Expression Bacteria Downregulation Gene Regulation Microbial Response Environmental Stress Molecular Biology Proteomics Stress Tolerance Cellular Adaptation Enzyme Activity Metabolic Pathways"
268	"Cold exposure increases BAT recruitment. Cold exposure increases BAT recruitment. Cold exposure increases BAT recruitment. Cold exposure increases BAT recruitment. Cold exposure increases BAT recruitment. Certainly! Here is a list of keywords for the query ""Cold exposure increases BAT recruitment"" Cold Exposure Brown Adipose Tissue (BAT) BAT Recruitment Thermogenesis Metabolism Cold-Induced Thermogenesis Adipose Tissue Energy Expenditure Sympathetic Nervous System Mitochondria UCP1 (Uncoupling Protein 1) Beige Adipocytes Cold Acclimation Body Temperature"
1245	The one-child policy has been successful in lowering population growth. The one-child policy has been successful in lowering population growth. The one-child policy has been successful in lowering population growth. The one-child policy has been successful in lowering population growth. The one-child policy has been successful in lowering population growth. One-child policy Population control Demographic trends Birth rates Fertility rates Family planning China Population growth Government policy Societal impacts Economic effects Environmental benefits Historical context Policy outcomes Long-term effects
148	Autophagy declines in aged organisms. Autophagy declines in aged organisms. Autophagy declines in aged organisms. Autophagy declines in aged organisms. Autophagy declines in aged organisms. Autophagy Age Aged Organisms Decline Cellular aging Degradation Lysosomes Protein turnover Senescence
269	"Cold exposure reduces BAT recruitment. Cold exposure reduces BAT recruitment. Cold exposure reduces BAT recruitment. Cold exposure reduces BAT recruitment. Cold exposure reduces BAT recruitment. Certainly! Here is a list of keywords for the query ""Cold exposure reduces BAT recruitment"" Cold exposure BAT (Brown Adipose Tissue) Recruitment Thermogenesis Metabolism Thermoregulation Adipocytes Cold-induced Energy expenditure Obesity Body temperature Sympathetic nervous system Mitochondria UCP1 (U uncoupling protein 1) Shivering Non-shivering therm"
820	N-terminal cleavage increases success identifying transcription start sites. N-terminal cleavage increases success identifying transcription start sites. N-terminal cleavage increases success identifying transcription start sites. N-terminal cleavage increases success identifying transcription start sites. N-terminal cleavage increases success identifying transcription start sites. N-terminal Cleavage Transcription Start Sites Identification Success Protein Sequencing Bioinformatics Gene Expression
700	Localization of PIN1 in the Arabidopsis embryo does not require VPS9a Localization of PIN1 in the Arabidopsis embryo does not require VPS9a Localization of PIN1 in the Arabidopsis embryo does not require VPS9a Localization of PIN1 in the Arabidopsis embryo does not require VPS9a Localization of PIN1 in the Arabidopsis embryo does not require VPS9a PIN1 Arabidopsis Embryo Localization VPS9a Protein trafficking Development Plant biology Cell polarity Auxin transport Vesicle trafficking Endocytosis Membrane proteins Plant embryos Genetic analysis
821	N-terminal cleavage reduces success identifying transcription start sites. N-terminal cleavage reduces success identifying transcription start sites. N-terminal cleavage reduces success identifying transcription start sites. N-terminal cleavage reduces success identifying transcription start sites. N-terminal cleavage reduces success identifying transcription start sites. N-terminal Cleavage Success Identifying Transcription Start Sites Protein Sequencing Bioinformatics Gene Expression Regulatory Analysis Enzymatic Degradation Mass Spectrometry RNA Promoter
702	Localization of PIN1 in the roots of Arabidopsis does not require VPS9a Localization of PIN1 in the roots of Arabidopsis does not require VPS9a Localization of PIN1 in the roots of Arabidopsis does not require VPS9a Localization of PIN1 in the roots of Arabidopsis does not require VPS9a Localization of PIN1 in the roots of Arabidopsis does not require VPS9a Localization PIN1 Roots Arabidopsis VPS9a Requirement Protein trafficking Plant biology Auxin transport Endocytosis Vesicle sorting Cellular distribution Gene expression Developmental biology Membrane proteins Vacuolar protein sorting Cellular mechanisms Molecular biology Plant roots PIN protein family
823	N348I mutations cause resistance to zidovudine (AZT). N348I mutations cause resistance to zidovudine (AZT). N348I mutations cause resistance to zidovudine (AZT). N348I mutations cause resistance to zidovudine (AZT). N348I mutations cause resistance to zidovudine (AZT). N348I Mutations Resistance Zidovudine AZT HIV Retrovirus Treatment Antiretroviral Drug Therapy Enzyme Reverse Transcriptase
42	A high microerythrocyte count raises vulnerability to severe anemia in homozygous alpha (+)- thalassemia trait subjects. A high microerythrocyte count raises vulnerability to severe anemia in homozygous alpha (+)- thalassemia trait subjects. A high microerythrocyte count raises vulnerability to severe anemia in homozygous alpha (+)- thalassemia trait subjects. A high microerythrocyte count raises vulnerability to severe anemia in homozygous alpha (+)- thalassemia trait subjects. A high microerythrocyte count raises vulnerability to severe anemia in homozygous alpha (+)- thalassemia trait subjects. Microerythrocyte High microerythrocyte count Severe anemia Homozygous Alpha thalassemia Alpha (+)-thalassemia trait Vulnerability Hemoglobin Red blood cells Genetic disorder Thalassemia Anemia Blood disorder Clinical significance Hematological trait
48	A total of 1,000 people in the UK are asymptomatic carriers of vCJD infection. A total of 1,000 people in the UK are asymptomatic carriers of vCJD infection. A total of 1,000 people in the UK are asymptomatic carriers of vCJD infection. A total of 1,000 people in the UK are asymptomatic carriers of vCJD infection. A total of 1,000 people in the UK are asymptomatic carriers of vCJD infection. 000 people UK asymptomatic carriers vCJD infection
49	"ADAR1 binds to Dicer to cleave pre-miRNA. ADAR1 binds to Dicer to cleave pre-miRNA. ADAR1 binds to Dicer to cleave pre-miRNA. ADAR1 binds to Dicer to cleave pre-miRNA. ADAR1 binds to Dicer to cleave pre-miRNA. Certainly! Here is a list of keywords for the query ""ADAR1 binds to Dicer to cleave pre-miRNA"" ADAR1 Dicer pre-miRNA Binding Cleavage RNA editing MicroRNA Ribonuclease Gene regulation RNA processing ADAR1-Dicer interaction Pre-miRNA processing RNA interference Non-coding RNA Post-transcriptional regulation"
1385	"cSMAC formation enhances weak ligand signalling. cSMAC formation enhances weak ligand signalling. cSMAC formation enhances weak ligand signalling. cSMAC formation enhances weak ligand signalling. cSMAC formation enhances weak ligand signalling. Certainly! Here is a list of keywords for the query ""cSMAC formation enhances weak ligand signalling"" cSMAC Central Supramolecular Activation Cluster Formation Enhances Weak Ligand Signalling Immunological Synapse T-cell Receptor Signal Transduction Cell Activation Ligand Binding Signaling Pathways Immune Response Cytoskeleton Protein Aggregation Molecular Mechanisms"
1021	Rapid up-regulation and higher basal expression of interferon-induced genes reduce survival of granule cell neurons that are infected by West Nile virus. Rapid up-regulation and higher basal expression of interferon-induced genes reduce survival of granule cell neurons that are infected by West Nile virus. Rapid up-regulation and higher basal expression of interferon-induced genes reduce survival of granule cell neurons that are infected by West Nile virus. Rapid up-regulation and higher basal expression of interferon-induced genes reduce survival of granule cell neurons that are infected by West Nile virus. Rapid up-regulation and higher basal expression of interferon-induced genes reduce survival of granule cell neurons that are infected by West Nile virus. Certainly! Here is a list of keywords for the provided query Rapid up-regulation Higher basal expression Interferon-induced genes Survival reduction Granule cell neurons West Nile virus Infection Gene expression Neurodegeneration Viral pathology
1020	Rapid up-regulation and higher basal expression of interferon-induced genes increase survival of granule cell neurons that are infected by West Nile virus. Rapid up-regulation and higher basal expression of interferon-induced genes increase survival of granule cell neurons that are infected by West Nile virus. Rapid up-regulation and higher basal expression of interferon-induced genes increase survival of granule cell neurons that are infected by West Nile virus. Rapid up-regulation and higher basal expression of interferon-induced genes increase survival of granule cell neurons that are infected by West Nile virus. Rapid up-regulation and higher basal expression of interferon-induced genes increase survival of granule cell neurons that are infected by West Nile virus. Rapid up-regulation Higher basal expression Interferon-induced genes Survival Granule cell neurons West Nile virus Infection Gene expression Neuroprotection Viral infection Cellular response Immune response Neurotropic viruses Molecular mechanisms Gene regulation
1262	The repair of Cas9-induced double strand breaks in human DNA is error-prone. The repair of Cas9-induced double strand breaks in human DNA is error-prone. The repair of Cas9-induced double strand breaks in human DNA is error-prone. The repair of Cas9-induced double strand breaks in human DNA is error-prone. The repair of Cas9-induced double strand breaks in human DNA is error-prone. Cas9 Double strand breaks Human DNA Repair Error-prone CRISPR Genomic instability Mutation NHEJ (Non-homologous end joining) Genetic editing DNA repair mechanisms Off-target effects Molecular biology Biotechnology Gene therapy
1140	Taking 400mg of α-tocopheryl acetate helps to prevent prostate cancer. Taking 400mg of α-tocopheryl acetate helps to prevent prostate cancer. Taking 400mg of α-tocopheryl acetate helps to prevent prostate cancer. Taking 400mg of α-tocopheryl acetate helps to prevent prostate cancer. Taking 400mg of α-tocopheryl acetate helps to prevent prostate cancer. 400mg α-tocopheryl acetate Vitamin E Prostate cancer Prevention Supplement Antioxidant Risk reduction Health benefit Clinical trial Study Research Male health Dosage Treatment Cancer prevention Nutritional supplement Biological activity Prostate health Medical research
1382	"aPKCz causes tumour enhancement by affecting glutamine metabolism. aPKCz causes tumour enhancement by affecting glutamine metabolism. aPKCz causes tumour enhancement by affecting glutamine metabolism. aPKCz causes tumour enhancement by affecting glutamine metabolism. aPKCz causes tumour enhancement by affecting glutamine metabolism. Sure! Here is a list of keywords for the query: ""aPKCz causes tumour enhancement by affecting glutamine metabolism aPKCz Tumour Enhancement Glutamine Metabolism Cancer Amino Acid Signaling Pathway Oncogenesis Cellular Metabolism Tumor Growth Glutamine Metabolism Kinase Proteins Pathophysiology Biological Mechanisms"
274	"Combination nicotine replacement therapies with varenicline or bupropion lead to significantly higher long-term abstinence rates at 52 weeks than varenicline monotherapy. Combination nicotine replacement therapies with varenicline or bupropion lead to significantly higher long-term abstinence rates at 52 weeks than varenicline monotherapy. Combination nicotine replacement therapies with varenicline or bupropion lead to significantly higher long-term abstinence rates at 52 weeks than varenicline monotherapy. Combination nicotine replacement therapies with varenicline or bupropion lead to significantly higher long-term abstinence rates at 52 weeks than varenicline monotherapy. Combination nicotine replacement therapies with varenicline or bupropion lead to significantly higher long-term abstinence rates at 52 weeks than varenicline monotherapy. Combination therapy Nicotine replacement therapy Varenicline Bupropion Long-term abstinence 52 weeks Smoking cessation Monotherapy Clinical trials 烟草依赖 (Note: This is ""Tobacco dependence"" in Chinese. If you prefer it in English it would be ""Tobacco dependence"" or ""Nicotine dependence"") Would you like the last keyword in English instead"
1019	Rapid phosphotransfer rates govern fidelity in two component systems Rapid phosphotransfer rates govern fidelity in two component systems Rapid phosphotransfer rates govern fidelity in two component systems Rapid phosphotransfer rates govern fidelity in two component systems Rapid phosphotransfer rates govern fidelity in two component systems Rapid phosphotransfer Fidelity Two component systems Signaling Kinase Response regulator Bacterial signaling Molecular mechanisms Regulation Phosphorylation Enzyme kinetics Signal transduction Protein interactions Microbiology Biochemistry
275	Combining phosphatidylinositide 3-kinase and MEK 1/2 inhibitors is effective at treating KRAS mutant tumors. Combining phosphatidylinositide 3-kinase and MEK 1/2 inhibitors is effective at treating KRAS mutant tumors. Combining phosphatidylinositide 3-kinase and MEK 1/2 inhibitors is effective at treating KRAS mutant tumors. Combining phosphatidylinositide 3-kinase and MEK 1/2 inhibitors is effective at treating KRAS mutant tumors. Combining phosphatidylinositide 3-kinase and MEK 1/2 inhibitors is effective at treating KRAS mutant tumors. Phosphatidylinositide 3-kinase MEK 1/2 inhibitors KRAS mutant tumors Combination therapy Cancer treatment Targeted therapy Signaling pathways Oncology Tumor inhibition Clinical efficacy
1259	The relationship between a breast cancer patient's capacity to metabolize tamoxifen and treatment outcome is dependent on the patient's genetic make-up. The relationship between a breast cancer patient's capacity to metabolize tamoxifen and treatment outcome is dependent on the patient's genetic make-up. The relationship between a breast cancer patient's capacity to metabolize tamoxifen and treatment outcome is dependent on the patient's genetic make-up. The relationship between a breast cancer patient's capacity to metabolize tamoxifen and treatment outcome is dependent on the patient's genetic make-up. The relationship between a breast cancer patient's capacity to metabolize tamoxifen and treatment outcome is dependent on the patient's genetic make-up. Certainly! Here is a list of keywords for the given query Breast cancer Tamoxifen Metabolism Treatment outcome Genetics Genetic make-up Patient Capacity Pharmacogenomics Enzyme activity CYP2D6 Biomarkers Genetic polymorphisms Personalized medicine Drug efficacy Adverse reactions Therapeutic response Biomolecular interactions Gen
1137	TNFAIP3 is a tumor suppressor in glioblastoma. TNFAIP3 is a tumor suppressor in glioblastoma. TNFAIP3 is a tumor suppressor in glioblastoma. TNFAIP3 is a tumor suppressor in glioblastoma. TNFAIP3 is a tumor suppressor in glioblastoma. TNFAIP3 Tumor suppressor Glioblastoma Cancer gene Oncology Neuro-oncology A20 (TNFAIP3 protein name) Gene expression Tumor progression Glioma Molecular biology Cellular regulation Inflammation NF-κB pathway Apoptosis Treatment Biomarker Prognosis Therapeutic
1379	Women with a higher birth weight are more likely to develop breast cancer later in life. Women with a higher birth weight are more likely to develop breast cancer later in life. Women with a higher birth weight are more likely to develop breast cancer later in life. Women with a higher birth weight are more likely to develop breast cancer later in life. Women with a higher birth weight are more likely to develop breast cancer later in life. Women Higher birth weight Breast cancer Development Later in life Risk factors Long-term health Epidemiology
399	Exposure to fine particulate air pollution is relate to anxiety prevalence. Exposure to fine particulate air pollution is relate to anxiety prevalence. Exposure to fine particulate air pollution is relate to anxiety prevalence. Exposure to fine particulate air pollution is relate to anxiety prevalence. Exposure to fine particulate air pollution is relate to anxiety prevalence. fine particulate matter air pollution anxiety prevalence exposure environmental health mental health particulate matter 2.5 (PM2.5) public health epidemiology air quality respiratory health urban environments correlation risk factors stress psychological impact long-term exposure short-term exposure health outcomes
279	Commelina yellow mottle virus' (ComYMV) genome consists of 7489 baise pairs. Commelina yellow mottle virus' (ComYMV) genome consists of 7489 baise pairs. Commelina yellow mottle virus' (ComYMV) genome consists of 7489 baise pairs. Commelina yellow mottle virus' (ComYMV) genome consists of 7489 baise pairs. Commelina yellow mottle virus' (ComYMV) genome consists of 7489 baise pairs. Commelina yellow mottle virus ComYMV Genome Baise pairs
1014	Rapamycin decreases the concentration of triacylglycerols in fruit flies. Rapamycin decreases the concentration of triacylglycerols in fruit flies. Rapamycin decreases the concentration of triacylglycerols in fruit flies. Rapamycin decreases the concentration of triacylglycerols in fruit flies. Rapamycin decreases the concentration of triacylglycerols in fruit flies. Rapamycin Triacylglycerols Fruit flies Concentration reduction Metabolic effect Lipid metabolism Insect biochemistry Dietary intervention Pharmacological effect Model organisms
830	NF2 (Merlin) causes phosphorylation and subsequent cytoplasmic sequestration of YAP in Drosophila by activating LATS1/2 kinases. NF2 (Merlin) causes phosphorylation and subsequent cytoplasmic sequestration of YAP in Drosophila by activating LATS1/2 kinases. NF2 (Merlin) causes phosphorylation and subsequent cytoplasmic sequestration of YAP in Drosophila by activating LATS1/2 kinases. NF2 (Merlin) causes phosphorylation and subsequent cytoplasmic sequestration of YAP in Drosophila by activating LATS1/2 kinases. NF2 (Merlin) causes phosphorylation and subsequent cytoplasmic sequestration of YAP in Drosophila by activating LATS1/2 kinases. Certainly! Here is a list of keywords for the query NF2 Merlin Phosphorylation YAP Cytoplasmic Sequestration Drosophila LATS1 LATS2 Kinases Activating Signal Transduction Hippo Pathway
831	NF2 (Merlin) prevents phosphorylation and subsequent cytoplasmic sequestration of YAP in Drosophila. NF2 (Merlin) prevents phosphorylation and subsequent cytoplasmic sequestration of YAP in Drosophila. NF2 (Merlin) prevents phosphorylation and subsequent cytoplasmic sequestration of YAP in Drosophila. NF2 (Merlin) prevents phosphorylation and subsequent cytoplasmic sequestration of YAP in Drosophila. NF2 (Merlin) prevents phosphorylation and subsequent cytoplasmic sequestration of YAP in Drosophila. NF2 Merlin Phosphorylation Cytoplasmic sequestration YAP Drosophila Tumor suppressor Hippo pathway Cell growth Development Gene regulation Protein interaction Signaling pathway Oncogenesis Tissue homeostasis
1012	"Radioiodine treatment of non-toxic multinodular goitre reduces thyroid volume. Radioiodine treatment of non-toxic multinodular goitre reduces thyroid volume. Radioiodine treatment of non-toxic multinodular goitre reduces thyroid volume. Radioiodine treatment of non-toxic multinodular goitre reduces thyroid volume. Radioiodine treatment of non-toxic multinodular goitre reduces thyroid volume. Certainly here is a list of keywords that are relevant to the query ""Radioiodine treatment of non-toxic multinodular goitre reduces thyroid volume"" Radioiodine Treatment Non-toxic Multinodular Goitre Thyroid Volume Reduction Iodine-131 Hyperthyroidism Thyroid Nodules Endocrine Radiotherapy Thyroid Function Clinical Trial"
832	"NFAT4 activation requires IP3R-mediated Ca2+ mobilization. NFAT4 activation requires IP3R-mediated Ca2+ mobilization. NFAT4 activation requires IP3R-mediated Ca2+ mobilization. NFAT4 activation requires IP3R-mediated Ca2+ mobilization. NFAT4 activation requires IP3R-mediated Ca2+ mobilization. Certainly! Here is a list of keywords for the query ""NFAT4 activation requires IP3R-mediated Ca2+ mobilization"" NFAT4 Activation IP3R IP3 Receptor Ca2+ Calcium Mobilization Signaling Cellular Pathway Regulation Intracellular Second Messenger Gene Expression Signaling Cascade These keywords cover the main components and processes involved in the query"
834	NOX2-independent pathways can generate peroxynitrite by reacting with nitrogen intermediates. NOX2-independent pathways can generate peroxynitrite by reacting with nitrogen intermediates. NOX2-independent pathways can generate peroxynitrite by reacting with nitrogen intermediates. NOX2-independent pathways can generate peroxynitrite by reacting with nitrogen intermediates. NOX2-independent pathways can generate peroxynitrite by reacting with nitrogen intermediates. Certainly! Here is a list of keywords for the given query NOX2-independent Pathways Peroxynitrite Generation Nitrogen Intermediates Reactive Oxidative Nitrosative Stress Biochemistry Enzymes Redox Signaling Molecular Mechanisms Oxidants Nitric Oxide
956	"Pleiotropic coupling of GLP-1R to intracellular effectors promotes distinct profiles of cellular signaling. Pleiotropic coupling of GLP-1R to intracellular effectors promotes distinct profiles of cellular signaling. Pleiotropic coupling of GLP-1R to intracellular effectors promotes distinct profiles of cellular signaling. Pleiotropic coupling of GLP-1R to intracellular effectors promotes distinct profiles of cellular signaling. Pleiotropic coupling of GLP-1R to intracellular effectors promotes distinct profiles of cellular signaling. Certainly! Here is a list of keywords for the query: ""P zupełnie coupling of GLP-1R to intracellular effectors promotes distinct profiles of cellular signaling Pleiotropic Coupling GLP-1R (Glucagon-like Peptide-1 Receptor) Intracellular Effectors Promotes Distinct Profiles Cellular Signaling These keywords capture the main concepts and elements of the query which focuses on the diverse and multiple effects of the GLP-1R receptor when it"
50	AIRE is expressed in some skin tumors. AIRE is expressed in some skin tumors. AIRE is expressed in some skin tumors. AIRE is expressed in some skin tumors. AIRE is expressed in some skin tumors. AIRE Skin Tumors Expression Antigen-Presenting Immune Regulation Cancer Biology Tumor Microenvironment Autoimmune Regulator Protein
715	Low expression of miR7a does represses target genes and exerts a biological function in ovaries. Low expression of miR7a does represses target genes and exerts a biological function in ovaries. Low expression of miR7a does represses target genes and exerts a biological function in ovaries. Low expression of miR7a does represses target genes and exerts a biological function in ovaries. Low expression of miR7a does represses target genes and exerts a biological function in ovaries. Certainly! Here is a list of keywords for the given query miR7a Low expression Represses Target genes Biological function Ovaries MicroRNA Gene regulation Ovarian biology Gene expression miRNA function Repression mechanism Ovarian genes miR7a target Molecular biology
957	Podocytes are motile and migrate in the presence of injury. Podocytes are motile and migrate in the presence of injury. Podocytes are motile and migrate in the presence of injury. Podocytes are motile and migrate in the presence of injury. Podocytes are motile and migrate in the presence of injury. Podocytes Motile Migration Injury Cellular movement Kidney disease Glomerular damage
51	"ALDH1 expression is associated with better breast cancer outcomes. ALDH1 expression is associated with better breast cancer outcomes. ALDH1 expression is associated with better breast cancer outcomes. ALDH1 expression is associated with better breast cancer outcomes. ALDH1 expression is associated with better breast cancer outcomes. Certainly! Here are the keywords for the query ""ALDH1 expression is associated with better breast cancer outcomes"" ALDH1 Expression Association Breast Cancer Outcomes Prognosis Biomarker Survival Cancer Cells Tumor Biology Gene Expression Clinical Significance Oncology Therapy Response Patient Outcomes"
716	"Low expression of miR7a exerts a biological function in testis. Low expression of miR7a exerts a biological function in testis. Low expression of miR7a exerts a biological function in testis. Low expression of miR7a exerts a biological function in testis. Low expression of miR7a exerts a biological function in testis. Certainly! Here is a list of keywords for the query ""Low expression of miR7a exerts a biological function in testis"" miR7a Low expression Biological function Testis MicroRNA Gene regulation Reproductive biology Male fertility Spermatogenesis RNA expression Molecular biology Sperm development Gene expression Testicular function Regulation of gene expression"
837	NR5A2 is important in development of endometrial tissues. NR5A2 is important in development of endometrial tissues. NR5A2 is important in development of endometrial tissues. NR5A2 is important in development of endometrial tissues. NR5A2 is important in development of endometrial tissues. NR5A2 Development Endometrial tissues Gene expression Hormone regulation Reproductive system Tissue differentiation Embryonic development Uterine biology Cell proliferation Endometrium Fertility Molecular biology Signaling pathways Transcription factors
53	"ALDH1 expression is associated with poorer prognosis in breast cancer. ALDH1 expression is associated with poorer prognosis in breast cancer. ALDH1 expression is associated with poorer prognosis in breast cancer. ALDH1 expression is associated with poorer prognosis in breast cancer. ALDH1 expression is associated with poorer prognosis in breast cancer. Sure here are the keywords for the query ""ALDH1 expression is associated with poorer prognosis in breast cancer"" ALDH1 Expression Poorer Prognosis Breast Cancer Association Biomarker Oncology Tumor Prognostic Factor Clinical Outcome"
718	Low nucleosome occupancy correlates with low methylation levels across species. Low nucleosome occupancy correlates with low methylation levels across species. Low nucleosome occupancy correlates with low methylation levels across species. Low nucleosome occupancy correlates with low methylation levels across species. Low nucleosome occupancy correlates with low methylation levels across species. Nucleosome Occupancy Methylation Levels Correlation Species Epigenetics Chromatin Genomics DNA Modification Gene Regulation Evolution Bioinformatics Molecular Biology
839	Nanoparticles can be targeted against specific cell types by incorporating aptamers into lipid nanoparticles. Nanoparticles can be targeted against specific cell types by incorporating aptamers into lipid nanoparticles. Nanoparticles can be targeted against specific cell types by incorporating aptamers into lipid nanoparticles. Nanoparticles can be targeted against specific cell types by incorporating aptamers into lipid nanoparticles. Nanoparticles can be targeted against specific cell types by incorporating aptamers into lipid nanoparticles. Nanoparticles Targeted delivery Cell types Aptamers Lipid nanoparticles Specificity Drug delivery Therapeutic applications Molecular recognition Biomolecular engineering
54	AMP-activated protein kinase (AMPK) activation increases inflammation-related fibrosis in the lungs. AMP-activated protein kinase (AMPK) activation increases inflammation-related fibrosis in the lungs. AMP-activated protein kinase (AMPK) activation increases inflammation-related fibrosis in the lungs. AMP-activated protein kinase (AMPK) activation increases inflammation-related fibrosis in the lungs. AMP-activated protein kinase (AMPK) activation increases inflammation-related fibrosis in the lungs. AMP-activated protein kinase (AMPK) AMPK activation Inflammation Fibrosis Lungs Pulmonary fibrosis Inflammation-related fibrosis AMPK and lung disease AMPK signaling Inflammatory pathways Fibrotic response Lung inflammation Protein kinases Cellular signaling Pathophysiology of fibrosis
56	APOE4 expression in iPSC-derived neurons increases AlphaBeta production and tau phosphorylation causing GABA neuron degeneration. APOE4 expression in iPSC-derived neurons increases AlphaBeta production and tau phosphorylation causing GABA neuron degeneration. APOE4 expression in iPSC-derived neurons increases AlphaBeta production and tau phosphorylation causing GABA neuron degeneration. APOE4 expression in iPSC-derived neurons increases AlphaBeta production and tau phosphorylation causing GABA neuron degeneration. APOE4 expression in iPSC-derived neurons increases AlphaBeta production and tau phosphorylation causing GABA neuron degeneration. APOE4 iPSC-derived neurons AlphaBeta production tau phosphorylation GABA neurons neuronal degeneration expression Alzheimer's disease neurodegeneration genetic risk factors
57	APOE4 expression in iPSC-derived neurons increases AlphaBeta production and tau phosphorylation, delaying GABA neuron degeneration. APOE4 expression in iPSC-derived neurons increases AlphaBeta production and tau phosphorylation, delaying GABA neuron degeneration. APOE4 expression in iPSC-derived neurons increases AlphaBeta production and tau phosphorylation, delaying GABA neuron degeneration. APOE4 expression in iPSC-derived neurons increases AlphaBeta production and tau phosphorylation, delaying GABA neuron degeneration. APOE4 expression in iPSC-derived neurons increases AlphaBeta production and tau phosphorylation, delaying GABA neuron degeneration. APOE4 iPSC-derived neurons AlphaBeta production Tau phosphorylation GABA neurons Neuron degeneration Expression Delay Neurodegeneration Alzheimer's disease Biomarkers Cellular models Neurobiology Protein expression Synaptic function
1274	The tip of the inner tube of the toxic type VI secretion system (T6SS) antibacterial effector in Escherichia coli (E. coli) carries toxic effector proteins. The tip of the inner tube of the toxic type VI secretion system (T6SS) antibacterial effector in Escherichia coli (E. coli) carries toxic effector proteins. The tip of the inner tube of the toxic type VI secretion system (T6SS) antibacterial effector in Escherichia coli (E. coli) carries toxic effector proteins. The tip of the inner tube of the toxic type VI secretion system (T6SS) antibacterial effector in Escherichia coli (E. coli) carries toxic effector proteins. The tip of the inner tube of the toxic type VI secretion system (T6SS) antibacterial effector in Escherichia coli (E. coli) carries toxic effector proteins. Toxic Type VI Secretion System (T6SS) Inner Tube Antibacterial Effector E. coli Toxic Effector Proteins Tip Escherichia coli Secretion Mechanism Bacterial Defense Microbial Pathogenesis
1395	p16INK4A accumulation is  linked to an abnormal wound response caused by the microinvasive step of advanced Oral Potentially Malignant Lesions (OPMLs). p16INK4A accumulation is  linked to an abnormal wound response caused by the microinvasive step of advanced Oral Potentially Malignant Lesions (OPMLs). p16INK4A accumulation is  linked to an abnormal wound response caused by the microinvasive step of advanced Oral Potentially Malignant Lesions (OPMLs). p16INK4A accumulation is  linked to an abnormal wound response caused by the microinvasive step of advanced Oral Potentially Malignant Lesions (OPMLs). p16INK4A accumulation is  linked to an abnormal wound response caused by the microinvasive step of advanced Oral Potentially Malignant Lesions (OPMLs). Certainly! Here is a list of keywords derived from the query p16INK4A Accumulation Abnormal Wound Response Microinvasive Step Advanced Oral Potentially Malignant Lesions OPMLs
1273	The sliding activity of kinesin-8 protein Kip3 promotes bipolar spindle assembly. The sliding activity of kinesin-8 protein Kip3 promotes bipolar spindle assembly. The sliding activity of kinesin-8 protein Kip3 promotes bipolar spindle assembly. The sliding activity of kinesin-8 protein Kip3 promotes bipolar spindle assembly. The sliding activity of kinesin-8 protein Kip3 promotes bipolar spindle assembly. kinesin-8 Kip3 sliding activity bipolar spindle spindle assembly protein function mitosis cell division microtubules motor protein
1272	The single flash-evoked ERG b-wave is generated by activity of ON-bipolar cells. The single flash-evoked ERG b-wave is generated by activity of ON-bipolar cells. The single flash-evoked ERG b-wave is generated by activity of ON-bipolar cells. The single flash-evoked ERG b-wave is generated by activity of ON-bipolar cells. The single flash-evoked ERG b-wave is generated by activity of ON-bipolar cells. Flash-evoked ERG B-wave ON-bipolar cells Retinal activity Electroretinogram Visual response Photoreceptors Bipolar cell activity Retinal function
1150	Tetraspanin-3 is a causative factor in the development of acute myelogenous leukemia Tetraspanin-3 is a causative factor in the development of acute myelogenous leukemia Tetraspanin-3 is a causative factor in the development of acute myelogenous leukemia Tetraspanin-3 is a causative factor in the development of acute myelogenous leukemia Tetraspanin-3 is a causative factor in the development of acute myelogenous leukemia Tetraspanin-3 Acute Myelogenous Leukemia Causative Factor Leukemia Development Tetraspanin-3 Function Myeloid Cells Hematopoietic System Oncogenesis Cancer Biomarker Leukemogenesis Molecular Mechanisms Genetic Factors Protein Expression Cellular Signaling Disease Pathology Biomolecular Interactions Treatment Targets
1271	The severity of cardiac involvement in amyloidosis can be described by the degree of transmurality of late gadolinium enhancement in MRI. The severity of cardiac involvement in amyloidosis can be described by the degree of transmurality of late gadolinium enhancement in MRI. The severity of cardiac involvement in amyloidosis can be described by the degree of transmurality of late gadolinium enhancement in MRI. The severity of cardiac involvement in amyloidosis can be described by the degree of transmurality of late gadolinium enhancement in MRI. The severity of cardiac involvement in amyloidosis can be described by the degree of transmurality of late gadolinium enhancement in MRI. Cardiac involvement Amyloidosis Severity Transmurality Late gadolinium enhancement MRI Cardiac MRI Amyloid cardiac disease Gadolinium Myocardial involvement Imaging Diagnostic imaging Cardiac imaging Myocardial enhancement Gadolinium enhancement
1270	The risk of male prisoners harming themselves is ten times that of female prisoners. The risk of male prisoners harming themselves is ten times that of female prisoners. The risk of male prisoners harming themselves is ten times that of female prisoners. The risk of male prisoners harming themselves is ten times that of female prisoners. The risk of male prisoners harming themselves is ten times that of female prisoners. Male prisoners Female prisoners Self-harm Risk Comparison Prison Statistics Gender differences Incidence Safety Mental health Suicide Risk factors inmate behavior Correctional facilities
163	Bariatric surgery has a positive impact on mental health. Bariatric surgery has a positive impact on mental health. Bariatric surgery has a positive impact on mental health. Bariatric surgery has a positive impact on mental health. Bariatric surgery has a positive impact on mental health. Bariatric surgery Mental health Psychological benefits Weight loss Depression Anxiety Self-esteem Quality of life Mood improvement Body image Eating disorders Cognitive function Stress reduction Emotional well-being Post-surgical outcomes
1029	Reduced responsiveness to interleukin-2 in regulatory T cells is associated with greater resistance to autoimmune diseases such as Type 1 Diabetes. Reduced responsiveness to interleukin-2 in regulatory T cells is associated with greater resistance to autoimmune diseases such as Type 1 Diabetes. Reduced responsiveness to interleukin-2 in regulatory T cells is associated with greater resistance to autoimmune diseases such as Type 1 Diabetes. Reduced responsiveness to interleukin-2 in regulatory T cells is associated with greater resistance to autoimmune diseases such as Type 1 Diabetes. Reduced responsiveness to interleukin-2 in regulatory T cells is associated with greater resistance to autoimmune diseases such as Type 1 Diabetes. Reduced responsiveness Interleukin-2 Regulatory T cells Autoimmune diseases Type 1 Diabetes Resistance Immune response Cytokine signaling T cell function Immunoregulation Autoimmunity Disease susceptibility Lymphocyte activation IL-2 signaling pathway Immune tolerance Therapeutic targets Genetic factors Immune regulation Pathogenesis
960	"Polymeal nutrition reduces cardiovascular mortality. Polymeal nutrition reduces cardiovascular mortality. Polymeal nutrition reduces cardiovascular mortality. Polymeal nutrition reduces cardiovascular mortality. Polymeal nutrition reduces cardiovascular mortality. Sure here are the keywords for the query ""Polymeal nutrition reduces cardiovascular mortality"" Polymeal Nutrition Cardiovascular Mortality Health benefits Diet Heart disease Risk reduction Food components Polyphenols Omega-3 fatty acids Fiber Antioxidants Blood pressure Cholesterol Clinical studies Epidemiology Prevention"
1389	mTORC2 regulates intracellular cysteine levels through xCT inhibition. mTORC2 regulates intracellular cysteine levels through xCT inhibition. mTORC2 regulates intracellular cysteine levels through xCT inhibition. mTORC2 regulates intracellular cysteine levels through xCT inhibition. mTORC2 regulates intracellular cysteine levels through xCT inhibition. mTORC2 Intracellular Cysteine Levels Regulation xCT Inhibition Amino Acid Cell Signaling Metabolic Pathway
1146	Teaching hospitals do not provide better care than non-teaching hospitals. Teaching hospitals do not provide better care than non-teaching hospitals. Teaching hospitals do not provide better care than non-teaching hospitals. Teaching hospitals do not provide better care than non-teaching hospitals. Teaching hospitals do not provide better care than non-teaching hospitals. Teaching hospitals Non-teaching hospitals Quality of care Patient outcomes Medical education Clinical performance Health care standards Physician training Hospital comparison Treatment effectiveness
1024	Recurrent mutations occur frequently within CTCF anchor sites adjacent to oncogenes. Recurrent mutations occur frequently within CTCF anchor sites adjacent to oncogenes. Recurrent mutations occur frequently within CTCF anchor sites adjacent to oncogenes. Recurrent mutations occur frequently within CTCF anchor sites adjacent to oncogenes. Recurrent mutations occur frequently within CTCF anchor sites adjacent to oncogenes. Recurrent mutations CTCF anchor sites Oncogenes Genetic mutations Cancer genetics Chromatin organization Transcriptional regulation Genomic instability Epigenetic modifiers Tumor suppressors DNA binding proteins Gene regulation Cancer genomics Mutation frequency Adjacent genes CTCF binding Genetic alterations Molecular biology Oncogenesis Cancer research
1266	The risk of breast cancer among parous women increases with placental weight of pregnancies, and this association is strongest for premenopausal breast cancer. The risk of breast cancer among parous women increases with placental weight of pregnancies, and this association is strongest for premenopausal breast cancer. The risk of breast cancer among parous women increases with placental weight of pregnancies, and this association is strongest for premenopausal breast cancer. The risk of breast cancer among parous women increases with placental weight of pregnancies, and this association is strongest for premenopausal breast cancer. The risk of breast cancer among parous women increases with placental weight of pregnancies, and this association is strongest for premenopausal breast cancer. Certainly! Here is a list of keywords for the given query Breast cancer Parous women Placental weight Pregnancies Premenopausal Cancer risk Association Obstetric factors Reproductive history Postpartum outcomes Hormonal influences Epidemiology Public health Medical research Cancer prevention
721	Lupus-prone mice infected with curliproducing bacteria have higher autoantibody titers compared to controls. Lupus-prone mice infected with curliproducing bacteria have higher autoantibody titers compared to controls. Lupus-prone mice infected with curliproducing bacteria have higher autoantibody titers compared to controls. Lupus-prone mice infected with curliproducing bacteria have higher autoantibody titers compared to controls. Lupus-prone mice infected with curliproducing bacteria have higher autoantibody titers compared to controls. Lupus-prone mice Curliproducing bacteria Autoantibody titers Infection Controls Lupus Autoimmunity Bacterial infection Antibody production Comparative study
1144	Taxation of sugar-sweetened beverages had no effect on the incidence rate of type II diabetes in India. Taxation of sugar-sweetened beverages had no effect on the incidence rate of type II diabetes in India. Taxation of sugar-sweetened beverages had no effect on the incidence rate of type II diabetes in India. Taxation of sugar-sweetened beverages had no effect on the incidence rate of type II diabetes in India. Taxation of sugar-sweetened beverages had no effect on the incidence rate of type II diabetes in India. Taxation Sugar-sweetened beverages SSB Type II diabetes Incidence rate India Public health Policy impact Beverage tax Health outcomes Non-communicable diseases Sugar tax Epidemiology Health economics Tax policy
723	Ly49Q directs the organization of neutrophil migration to inflammation sites by regulating membrane raft functions. Ly49Q directs the organization of neutrophil migration to inflammation sites by regulating membrane raft functions. Ly49Q directs the organization of neutrophil migration to inflammation sites by regulating membrane raft functions. Ly49Q directs the organization of neutrophil migration to inflammation sites by regulating membrane raft functions. Ly49Q directs the organization of neutrophil migration to inflammation sites by regulating membrane raft functions. Ly49Q Neutrophil Migration Inflammation Sites Membrane Rafts Regulation Organization Immune Response Cellular Trafficking Inflammatory Processes Signaling Pathways
845	"Neutrophil extracellular traps (NETs) are released by ANCA-stimulated neutrophils. Neutrophil extracellular traps (NETs) are released by ANCA-stimulated neutrophils. Neutrophil extracellular traps (NETs) are released by ANCA-stimulated neutrophils. Neutrophil extracellular traps (NETs) are released by ANCA-stimulated neutrophils. Neutrophil extracellular traps (NETs) are released by ANCA-stimulated neutrophils. Certainly! Here is a list of keywords for the query ""Neutrophil extracellular traps (NETs) are released by ANCA-stimulated neutrophils"" Neutrophil extracellular traps (NETs) ANCA (Antineutrophil cytoplasmic antibodies) Neutrophils Release Stimulation Immune response Inflammation Autoimmune diseases Microbe trapping Vascular damage Cell activation NETosis Pathogenesis"
967	Pretreatment with the Arp2/3 inhibitor CK-666 affects lamelliopodia formation. Pretreatment with the Arp2/3 inhibitor CK-666 affects lamelliopodia formation. Pretreatment with the Arp2/3 inhibitor CK-666 affects lamelliopodia formation. Pretreatment with the Arp2/3 inhibitor CK-666 affects lamelliopodia formation. Pretreatment with the Arp2/3 inhibitor CK-666 affects lamelliopodia formation. Pretreatment Arp2/3 Inhibitor CK-666 Lamelliopodia Formation Actin Cytoskeleton Cell Migration Cellular Dynamics Microscopy Cell Biology Protein Complex Filopodia Cell Morphology
847	New drugs for tuberculosis often do not penetrate the necrotic portion of a tuberculosis lesion in high concentrations. New drugs for tuberculosis often do not penetrate the necrotic portion of a tuberculosis lesion in high concentrations. New drugs for tuberculosis often do not penetrate the necrotic portion of a tuberculosis lesion in high concentrations. New drugs for tuberculosis often do not penetrate the necrotic portion of a tuberculosis lesion in high concentrations. New drugs for tuberculosis often do not penetrate the necrotic portion of a tuberculosis lesion in high concentrations. Tuberculosis New drugs Necrotic portion Lesion High concentrations Penetration Treatment Pharmacokinetics Granuloma Bacterial persistence
727	Ly6C hi monocytes have a lower inflammatory capacity compared to their Ly6C lo counterparts. Ly6C hi monocytes have a lower inflammatory capacity compared to their Ly6C lo counterparts. Ly6C hi monocytes have a lower inflammatory capacity compared to their Ly6C lo counterparts. Ly6C hi monocytes have a lower inflammatory capacity compared to their Ly6C lo counterparts. Ly6C hi monocytes have a lower inflammatory capacity compared to their Ly6C lo counterparts. Ly6C hi monocytes Ly6C lo monocytes Inflammatory capacity Comparison Monocytes Inflammation Immune response Cytokines Immune cells Phenotype Ly6C expression Macrophages Innate immunity Regulatory mechanisms Cellular activation
728	Ly6C hi monocytes have a lower inflammatory capacity than Ly6C lo monocytes. Ly6C hi monocytes have a lower inflammatory capacity than Ly6C lo monocytes. Ly6C hi monocytes have a lower inflammatory capacity than Ly6C lo monocytes. Ly6C hi monocytes have a lower inflammatory capacity than Ly6C lo monocytes. Ly6C hi monocytes have a lower inflammatory capacity than Ly6C lo monocytes. Ly6C Monocytes Inflammatory Capacity Ly6C hi Ly6C lo Immune Cells Inflammation Phenotype Response Cytokines Expression Macrophages Differentiation
729	"Lymphadenopathy is observed in knockin mouse lacking the SHP-2 MAPK pathway. Lymphadenopathy is observed in knockin mouse lacking the SHP-2 MAPK pathway. Lymphadenopathy is observed in knockin mouse lacking the SHP-2 MAPK pathway. Lymphadenopathy is observed in knockin mouse lacking the SHP-2 MAPK pathway. Lymphadenopathy is observed in knockin mouse lacking the SHP-2 MAPK pathway. Certainly! Here is a list of keywords for the query ""Lymphadenopathy is observed in knockin mouse lacking the SHP-2 MAPK pathway"" Lymphadenopathy Knockin mouse SHP-2 MAPK pathway Genetic modification Mouse model Immune system Lymph nodes Protein tyrosine phosphatase Signal transduction Pathophysiology Experimental biology Molecular genetics Disease mechanism Gene knockout"
1163	"The DdrB protein from Deinococcus radiodurans is an alternative SSB. The DdrB protein from Deinococcus radiodurans is an alternative SSB. The DdrB protein from Deinococcus radiodurans is an alternative SSB. The DdrB protein from Deinococcus radiodurans is an alternative SSB. The DdrB protein from Deinococcus radiodurans is an alternative SSB. Certainly! Here is a list of keywords for the query ""The DdrB protein from Deinococcus radiodurans is an alternative SSB"" DdrB Deinococcus radiodurans SSB Single-strand binding protein Alternative SSB Bacterial proteins Radiation resistance DNA repair Genomic stability Molecular biology Protein function Extremophile bacteria Microbial genetics Nucleic acid binding"
1041	Replacement of histone H2A with H2A.Z slows gene activation in yeasts by stabilizing +1 nucleosomes. Replacement of histone H2A with H2A.Z slows gene activation in yeasts by stabilizing +1 nucleosomes. Replacement of histone H2A with H2A.Z slows gene activation in yeasts by stabilizing +1 nucleosomes. Replacement of histone H2A with H2A.Z slows gene activation in yeasts by stabilizing +1 nucleosomes. Replacement of histone H2A with H2A.Z slows gene activation in yeasts by stabilizing +1 nucleosomes. Histone H2A H2A.Z Gene activation Yeasts Nucleosomes +1 nucleosomes Stabilization Chromatin structure Epigenetic regulation Transcriptional dynamics
171	Basophils counteract disease development in patients with systemic lupus erythematosus (SLE). Basophils counteract disease development in patients with systemic lupus erythematosus (SLE). Basophils counteract disease development in patients with systemic lupus erythematosus (SLE). Basophils counteract disease development in patients with systemic lupus erythematosus (SLE). Basophils counteract disease development in patients with systemic lupus erythematosus (SLE). Basophils Systemic Lupus Erythematosus (SLE) Disease Development Counteract Patients Immune Response Inflammation Autoimmune Disease Leukocytes Immunomodulation Cytokines Pathogenesis Therapy Prognosis Medical Research Clinical Studies Inflammatory Markers Immune Cells Lupus
1282	Therapeutic use of the drug Dapsone to treat pyoderma gangrenous is based on anecdotal evidence. Therapeutic use of the drug Dapsone to treat pyoderma gangrenous is based on anecdotal evidence. Therapeutic use of the drug Dapsone to treat pyoderma gangrenous is based on anecdotal evidence. Therapeutic use of the drug Dapsone to treat pyoderma gangrenous is based on anecdotal evidence. Therapeutic use of the drug Dapsone to treat pyoderma gangrenous is based on anecdotal evidence. Dapsone Pyoderma Gangrenosum Therapeutic Use Anecdotal Evidence Treatment Drug Therapy Skin Condition Inflammatory Disorder Off-Label Use Medical Case Studies
1281	"The ureABIEFGH gene cluster is induced by nickel (II) ion. The ureABIEFGH gene cluster is induced by nickel (II) ion. The ureABIEFGH gene cluster is induced by nickel (II) ion. The ureABIEFGH gene cluster is induced by nickel (II) ion. The ureABIEFGH gene cluster is induced by nickel (II) ion. Certainly! Here's a list of keywords related to the query ""The ureABIEFGH gene cluster is induced by nickel (II) ion"" UreABIEFGH Gene cluster Induction Nickel (II) ion Gene expression Metal ions Bacterial genetics Regulatory mechanisms Microbial metabolism Environmental response These keywords should help in researching and understanding the specific interaction between the ureABIEFGH gene cluster and nickel (II) ions"
294	Crossover hot spots are not found within gene promoters in Saccharomyces cerevisiae. Crossover hot spots are not found within gene promoters in Saccharomyces cerevisiae. Crossover hot spots are not found within gene promoters in Saccharomyces cerevisiae. Crossover hot spots are not found within gene promoters in Saccharomyces cerevisiae. Crossover hot spots are not found within gene promoters in Saccharomyces cerevisiae. Crossover Hot spots Gene promoters Saccharomyces cerevisiae Recombination Yeast genetics Meiosis Chromosomal regions Genetic mapping DNA recombination sites
1280	The ureABIEFGH gene cluster encodes urease maturation proteins : UreD/UreH, UreE, UreF, and UreG. The ureABIEFGH gene cluster encodes urease maturation proteins : UreD/UreH, UreE, UreF, and UreG. The ureABIEFGH gene cluster encodes urease maturation proteins : UreD/UreH, UreE, UreF, and UreG. The ureABIEFGH gene cluster encodes urease maturation proteins : UreD/UreH, UreE, UreF, and UreG. The ureABIEFGH gene cluster encodes urease maturation proteins : UreD/UreH, UreE, UreF, and UreG. UreABIEFGH Gene cluster Urease maturation proteins UreD UreH UreE UreF UreG Urease Protein maturation
295	Crosstalk between dendritic cells (DCs) and innate lymphoid cells (ILCs) is important in the regulation of intestinal homeostasis. Crosstalk between dendritic cells (DCs) and innate lymphoid cells (ILCs) is important in the regulation of intestinal homeostasis. Crosstalk between dendritic cells (DCs) and innate lymphoid cells (ILCs) is important in the regulation of intestinal homeostasis. Crosstalk between dendritic cells (DCs) and innate lymphoid cells (ILCs) is important in the regulation of intestinal homeostasis. Crosstalk between dendritic cells (DCs) and innate lymphoid cells (ILCs) is important in the regulation of intestinal homeostasis. Dendritic Cells (DCs) Innate Lymphoid Cells (ILCs) Crosstalk Intestinal Homeostasis Immune Regulation Gut Microbiota Inflammation Tissue Repair Mucosal Immunity Cytokines Signaling Pathways Immune Cells Interaction Barrier Function Host Defense Tolerance Intestinal Microenvironment Innate Immunity
298	Cytochrome c is released from the mitochondrial intermembrane space to cytosol during apoptosis. Cytochrome c is released from the mitochondrial intermembrane space to cytosol during apoptosis. Cytochrome c is released from the mitochondrial intermembrane space to cytosol during apoptosis. Cytochrome c is released from the mitochondrial intermembrane space to cytosol during apoptosis. Cytochrome c is released from the mitochondrial intermembrane space to cytosol during apoptosis. Cytochrome c Mitochondrial intermembrane space Cytosol Apoptosis Release Cell death Mitochondria Apoptotic signaling Caspase activation Intracellular signaling Membrane permeabilization Cellular response Biochemical pathway Protein translocation Cell cycle regulation
179	Birth-weight is positively associated with breast cancer. Birth-weight is positively associated with breast cancer. Birth-weight is positively associated with breast cancer. Birth-weight is positively associated with breast cancer. Birth-weight is positively associated with breast cancer. Birth-weight Breast cancer Positive association Correlation Risk factor Maternal health Neonatal characteristics Cancer epidemiology Long-term health outcomes Hormonal influences
971	Primary cervical cancer screening with HPV detection has higher longitudinal sensitivity than conventional cytology to detect cervical intraepithelial neoplasia grade 2. Primary cervical cancer screening with HPV detection has higher longitudinal sensitivity than conventional cytology to detect cervical intraepithelial neoplasia grade 2. Primary cervical cancer screening with HPV detection has higher longitudinal sensitivity than conventional cytology to detect cervical intraepithelial neoplasia grade 2. Primary cervical cancer screening with HPV detection has higher longitudinal sensitivity than conventional cytology to detect cervical intraepithelial neoplasia grade 2. Primary cervical cancer screening with HPV detection has higher longitudinal sensitivity than conventional cytology to detect cervical intraepithelial neoplasia grade 2. Cervical cancer screening HPV detection Longitudinal sensitivity Conventional cytology Cervical intraepithelial neoplasia CIN2 Primary screening Human Papillomavirus Cytological testing Cancer prevention Comparative effectiveness Gynecological oncology Precancerous lesions Screening methods Early detection
1279	The treatment of cancer patients with co-IR blockade precipitates adverse autoimmune events. The treatment of cancer patients with co-IR blockade precipitates adverse autoimmune events. The treatment of cancer patients with co-IR blockade precipitates adverse autoimmune events. The treatment of cancer patients with co-IR blockade precipitates adverse autoimmune events. The treatment of cancer patients with co-IR blockade precipitates adverse autoimmune events. Cancer treatment Co-IR blockade Adverse events Autoimmune reactions Immunotherapy Cancer patients Immune checkpoint inhibitors Co-inhibitory receptors Immune-related adverse events Autoimmunity Oncology Therapeutic antibodies Immune modulation Immune system Clinical outcomes
1278	The treatment of cancer patients with co-IR blockade does not cause any adverse autoimmune events. The treatment of cancer patients with co-IR blockade does not cause any adverse autoimmune events. The treatment of cancer patients with co-IR blockade does not cause any adverse autoimmune events. The treatment of cancer patients with co-IR blockade does not cause any adverse autoimmune events. The treatment of cancer patients with co-IR blockade does not cause any adverse autoimmune events. Cancer Treatment Co-IR Blockade Adverse Autoimmune Events Patients Immunotherapy Safety Clinical Trials Immunological Side Effects Tolerance Immune Response Therapy Co-Inhibitory
852	Non-invasive ventilation use should be decreased if there is inadequate response to conventional treatment. Non-invasive ventilation use should be decreased if there is inadequate response to conventional treatment. Non-invasive ventilation use should be decreased if there is inadequate response to conventional treatment. Non-invasive ventilation use should be decreased if there is inadequate response to conventional treatment. Non-invasive ventilation use should be decreased if there is inadequate response to conventional treatment. Non-invasive ventilation Inadequate response Conventional treatment Decrease use Treatment efficacy Clinical outcomes Respiratory therapy Ventilatory support Patient management Therapeutic intervention
975	Primary pro-inflammatory cytokines induce secondary pro- and anti-inflammatory mediators. Primary pro-inflammatory cytokines induce secondary pro- and anti-inflammatory mediators. Primary pro-inflammatory cytokines induce secondary pro- and anti-inflammatory mediators. Primary pro-inflammatory cytokines induce secondary pro- and anti-inflammatory mediators. Primary pro-inflammatory cytokines induce secondary pro- and anti-inflammatory mediators. Primary cytokines Pro-inflammatory cytokines Secondary mediators Pro-inflammatory mediators Anti-inflammatory mediators Cytokine induction Inflammatory response Immune response Cytokine signaling Inflammation regulation
613	Increased microtubule acetylation repairs LRRK2 Roc-COR domain mutation induced locomotor deficits. Increased microtubule acetylation repairs LRRK2 Roc-COR domain mutation induced locomotor deficits. Increased microtubule acetylation repairs LRRK2 Roc-COR domain mutation induced locomotor deficits. Increased microtubule acetylation repairs LRRK2 Roc-COR domain mutation induced locomotor deficits. Increased microtubule acetylation repairs LRRK2 Roc-COR domain mutation induced locomotor deficits. Microtubule acetylation LRRK2 Roc-COR domain Mutation Locomotor deficits Repair Neurodegeneration Parkinson's disease Kinase activity Cellular function Axonal transport Motor function Protein modification Therapeutic intervention Molecular mechanism
70	Activation of PPM1D suppresses p53 function. Activation of PPM1D suppresses p53 function. Activation of PPM1D suppresses p53 function. Activation of PPM1D suppresses p53 function. Activation of PPM1D suppresses p53 function. PPM1D Activation p53 Suppression Function Inhibition Tumor suppressor Phosphatase Cellular response Cancer Signaling pathways DNA damage Cell cycle Apoptosis Oncogenesis
72	Activator-inhibitor pairs are provided dorsally by Admpchordin. Activator-inhibitor pairs are provided dorsally by Admpchordin. Activator-inhibitor pairs are provided dorsally by Admpchordin. Activator-inhibitor pairs are provided dorsally by Admpchordin. Activator-inhibitor pairs are provided dorsally by Admpchordin. Activator-inhibitor Pairs Dorsally Admp Chordin Developmental biology Morphogens Signaling pathways Embryogenesis Gene expression Dorsal-ventral axis Cellular interaction Pattern formation Tissue differentiation Biochemical regulation
859	Normal expression of RUNX1 has tumor-promoting effects. Normal expression of RUNX1 has tumor-promoting effects. Normal expression of RUNX1 has tumor-promoting effects. Normal expression of RUNX1 has tumor-promoting effects. Normal expression of RUNX1 has tumor-promoting effects. RUNX1 Normal expression Tumor-promoting Oncogenesis Gene regulation Transcription factor Cancer Cellular proliferation Apoptosis Signaling pathways
619	Increased vessel density along with a reduction in fibrosis decreases the efficacy of chemotherapy treatments. Increased vessel density along with a reduction in fibrosis decreases the efficacy of chemotherapy treatments. Increased vessel density along with a reduction in fibrosis decreases the efficacy of chemotherapy treatments. Increased vessel density along with a reduction in fibrosis decreases the efficacy of chemotherapy treatments. Increased vessel density along with a reduction in fibrosis decreases the efficacy of chemotherapy treatments. Vessel density Fibrosis reduction Chemotherapy efficacy Tumor microenvironment Angiogenesis Blood vessel formation Tissue fibrosis Cancer treatment Drug delivery Tumor response Treatment resistance Oncology Vascular remodeling Hypoxia Tumor stroma Therapy outcome Medical oncology Cancer biology Vascular normalization Clinical trials
75	Active H. pylori urease has a polymeric structure that compromises two subunits, UreA and UreB. Active H. pylori urease has a polymeric structure that compromises two subunits, UreA and UreB. Active H. pylori urease has a polymeric structure that compromises two subunits, UreA and UreB. Active H. pylori urease has a polymeric structure that compromises two subunits, UreA and UreB. Active H. pylori urease has a polymeric structure that compromises two subunits, UreA and UreB. Active H. pylori Urease Polymeric Structure Subunits UreA UreB
1175	The PPR MDA5 has two N-terminal CARD domains. The PPR MDA5 has two N-terminal CARD domains. The PPR MDA5 has two N-terminal CARD domains. The PPR MDA5 has two N-terminal CARD domains. The PPR MDA5 has two N-terminal CARD domains. PPR MDA5 N-terminal CARD domains immune signaling protein recognition receptor
180	Blocking the interaction between TDP-43 and respiratory complex I proteins ND3 and ND6 leads to increased TDP-43-induced neuronal loss. Blocking the interaction between TDP-43 and respiratory complex I proteins ND3 and ND6 leads to increased TDP-43-induced neuronal loss. Blocking the interaction between TDP-43 and respiratory complex I proteins ND3 and ND6 leads to increased TDP-43-induced neuronal loss. Blocking the interaction between TDP-43 and respiratory complex I proteins ND3 and ND6 leads to increased TDP-43-induced neuronal loss. Blocking the interaction between TDP-43 and respiratory complex I proteins ND3 and ND6 leads to increased TDP-43-induced neuronal loss. TDP-43 Respiratory complex I ND3 ND6 Neuronal loss Interaction blocking Neurodegeneration Mitochondrial dysfunction Protein-protein interaction TDP-43 aggregation Neuronal death Neuroprotective strategies ALS (Amyotrophic Lateral Sclerosis) FTD (Frontotemporal Dementia) Neurotoxicity
183	Bone marrow cells contribute to adult macrophage compartments. Bone marrow cells contribute to adult macrophage compartments. Bone marrow cells contribute to adult macrophage compartments. Bone marrow cells contribute to adult macrophage compartments. Bone marrow cells contribute to adult macrophage compartments. Bone marrow Cells Contribution Adult Macrophages Compartments Hematopoiesis Monocytes Tissue-resident Differentiation Immune system Inflammation Homeostasis Microenvironment Circulating
1292	"There is no association between HNF4A mutations and diabetes risks. There is no association between HNF4A mutations and diabetes risks. There is no association between HNF4A mutations and diabetes risks. There is no association between HNF4A mutations and diabetes risks. There is no association between HNF4A mutations and diabetes risks. Certainly! Here is a list of keywords for the query ""There is no association between HNF4A mutations and diabetes risks"" HNF4A Mutations Diabetes Risks Association Genetic Disease Research Study Evidence Negative Correlation No Link Genetic Disorders Insulin Resistance Metabolic Disorders Type 2 Diabetes Type 1 Diabetes Genetic Factors"
185	Breast cancer development is determined exclusively by genetic factors. Breast cancer development is determined exclusively by genetic factors. Breast cancer development is determined exclusively by genetic factors. Breast cancer development is determined exclusively by genetic factors. Breast cancer development is determined exclusively by genetic factors. Breast cancer Genetic factors Exclusivity Development Heredity Genes Molecular genetics Oncogenes Tumor suppressor genes Inherited mutations BRCA1 BRCA2 Genetic predisposition Familial breast cancer Genetic testing
1290	There is an inverse relationship between hip fractures and statin use. There is an inverse relationship between hip fractures and statin use. There is an inverse relationship between hip fractures and statin use. There is an inverse relationship between hip fractures and statin use. There is an inverse relationship between hip fractures and statin use. Hip fractures Statin use Inverse relationship Statins Bone health Fracture risk Cholesterol-lowering drugs Osteoporosis Elderly patients Pharmacological intervention Clinical studies Epidemiological data Medication effects Public health Preventive medicine
1049	Ribosomopathies have a low degree of cell and tissue specific pathology. Ribosomopathies have a low degree of cell and tissue specific pathology. Ribosomopathies have a low degree of cell and tissue specific pathology. Ribosomopathies have a low degree of cell and tissue specific pathology. Ribosomopathies have a low degree of cell and tissue specific pathology. Ribosomopathies Cell-specific Tissue-specific Pathology Low degree Cellular Tissue Ribosomes Disease mechanism Genetic disorders
982	"Proteins synthesized at the growth cone are ubiquitinated at a higher rate than proteins from the cell body. Proteins synthesized at the growth cone are ubiquitinated at a higher rate than proteins from the cell body. Proteins synthesized at the growth cone are ubiquitinated at a higher rate than proteins from the cell body. Proteins synthesized at the growth cone are ubiquitinated at a higher rate than proteins from the cell body. Proteins synthesized at the growth cone are ubiquitinated at a higher rate than proteins from the cell body. Certainly! Here is a list of keywords for the query: ""Proteins synthesized at the growth cone are ubiquitinated at a higher rate than proteins from the cell body Proteins Synthesized Growth cone Ubiquitinated Higher rate Cell body Neurons Axon Proteasome Post-translational modification Protein degradation Local protein synthesis Neurodevelopment Signaling pathways Synaptic plasticity"
742	Macrolides have no protective effect against myocardial infarction. Macrolides have no protective effect against myocardial infarction. Macrolides have no protective effect against myocardial infarction. Macrolides have no protective effect against myocardial infarction. Macrolides have no protective effect against myocardial infarction. Macrolides Protective Effect Myocardial Infarction Cardiovascular Antibiotics Heart Attack Prevention Treatment Clinical Studies Research Outcomes
501	Headaches are not correlated with cognitive impairment. Headaches are not correlated with cognitive impairment. Headaches are not correlated with cognitive impairment. Headaches are not correlated with cognitive impairment. Headaches are not correlated with cognitive impairment. Headaches Cognitive impairment Correlation Neurology Brain function Pain Mental health Medical research Symptoms Neurocognitive disorders
743	"Macrolides protect against myocardial infarction. Macrolides protect against myocardial infarction. Macrolides protect against myocardial infarction. Macrolides protect against myocardial infarction. Macrolides protect against myocardial infarction. Certainly! Here is a list of keywords for the query ""Macrolides protect against myocardial infarction"" Macrolides Antibiotics Myocardial infarction Heart attack Cardioprotection Cardiovascular disease Anti-inflammatory Immune response Atherosclerosis Clinical trials Research studies Mechanisms of action Cardiovascular health Pharmacology Prevention Treatment Cardiology"
985	"Pseudogene PTENP1 regulates the expression of PTEN by functioning as an miRNA decoy. Pseudogene PTENP1 regulates the expression of PTEN by functioning as an miRNA decoy. Pseudogene PTENP1 regulates the expression of PTEN by functioning as an miRNA decoy. Pseudogene PTENP1 regulates the expression of PTEN by functioning as an miRNA decoy. Pseudogene PTENP1 regulates the expression of PTEN by functioning as an miRNA decoy. Certainly! Here is a list of keywords for the query ""Pseudogene PTENP1 regulates the expression of PTEN by functioning as an miRNA decoy"" Pseudogene PTENP1 PTEN Regulation Expression miRNA Decoy Gene Expression Non-coding RNA Competitive Endogenous RNA (ceRNA) miRNA Target mRNA Stability Tumor Suppressor Oncogene Gene Silencing"
502	Healthcare delivery efficiency in crowded delivery centers is impaired by improving structural, logistical, and interpersonal elements. Healthcare delivery efficiency in crowded delivery centers is impaired by improving structural, logistical, and interpersonal elements. Healthcare delivery efficiency in crowded delivery centers is impaired by improving structural, logistical, and interpersonal elements. Healthcare delivery efficiency in crowded delivery centers is impaired by improving structural, logistical, and interpersonal elements. Healthcare delivery efficiency in crowded delivery centers is impaired by improving structural, logistical, and interpersonal elements. Healthcare delivery Efficiency Crowded delivery centers Structural elements Logistical elements Interpersonal elements Improvement Healthcare systems Patient flow Resource management Staff coordination Clinical workflows Patient satisfaction Facility design Operational strategies Queue management Health services research Quality improvement Healthcare environment Patient care protocols
623	"Individuals with low serum vitamin D concentrations have increased risk of multiple sclerosis. Individuals with low serum vitamin D concentrations have increased risk of multiple sclerosis. Individuals with low serum vitamin D concentrations have increased risk of multiple sclerosis. Individuals with low serum vitamin D concentrations have increased risk of multiple sclerosis. Individuals with low serum vitamin D concentrations have increased risk of multiple sclerosis. Certainly! Here is a list of keywords for the query: ""Individuals with low serum vitamin D concentrations have increased risk of multiple sclerosis Low serum vitamin D Vitamin D deficiency Multiple sclerosis Risk factors Neurological disorders Epidemiology Autoimmune diseases Vitamin D status Serum concentrations Health outcomes"
744	Macropinocytosis contributes to a cell's supply of amino acids via the intracellular uptake of protein. Macropinocytosis contributes to a cell's supply of amino acids via the intracellular uptake of protein. Macropinocytosis contributes to a cell's supply of amino acids via the intracellular uptake of protein. Macropinocytosis contributes to a cell's supply of amino acids via the intracellular uptake of protein. Macropinocytosis contributes to a cell's supply of amino acids via the intracellular uptake of protein. Macropinocytosis Amino acids Intracellular uptake Protein Cell supply Endocytosis Nutrient acquisition Cellular metabolism Vesicle formation Cytoplasmic ingestion
507	Helminths interfere with immune system control of macrophages activated by IL-4 favor Mycobacterium tuberculosis replication. Helminths interfere with immune system control of macrophages activated by IL-4 favor Mycobacterium tuberculosis replication. Helminths interfere with immune system control of macrophages activated by IL-4 favor Mycobacterium tuberculosis replication. Helminths interfere with immune system control of macrophages activated by IL-4 favor Mycobacterium tuberculosis replication. Helminths interfere with immune system control of macrophages activated by IL-4 favor Mycobacterium tuberculosis replication. Helminths Immune system Macrophages IL-4 Mycobacterium tuberculosis Replication Interference Activation Pathogenesis Host-pathogen interaction
628	Infection of human T-cell lymphotropic virus type 1 is most frequent in individuals of African origin. Infection of human T-cell lymphotropic virus type 1 is most frequent in individuals of African origin. Infection of human T-cell lymphotropic virus type 1 is most frequent in individuals of African origin. Infection of human T-cell lymphotropic virus type 1 is most frequent in individuals of African origin. Infection of human T-cell lymphotropic virus type 1 is most frequent in individuals of African origin. Certainly! Here is a list of keywords for the query Infection Human T-cell lymphotropic virus HTLV-1 Frequency African origin Individuals Epidemiology Viral infection T-cell Lymphotropic
508	Hematopoietic Stem Cell purification reaches purity rate of up to 50%. Hematopoietic Stem Cell purification reaches purity rate of up to 50%. Hematopoietic Stem Cell purification reaches purity rate of up to 50%. Hematopoietic Stem Cell purification reaches purity rate of up to 50%. Hematopoietic Stem Cell purification reaches purity rate of up to 50%. Hematopoietic Stem Cell Purification Purity Rate 50%
1187	The YAP1 and TEAD complex tanslocates into the nucleus where it interacts with transcription factors and DNA-binding proteins that modulate target gene transcription. The YAP1 and TEAD complex tanslocates into the nucleus where it interacts with transcription factors and DNA-binding proteins that modulate target gene transcription. The YAP1 and TEAD complex tanslocates into the nucleus where it interacts with transcription factors and DNA-binding proteins that modulate target gene transcription. The YAP1 and TEAD complex tanslocates into the nucleus where it interacts with transcription factors and DNA-binding proteins that modulate target gene transcription. The YAP1 and TEAD complex tanslocates into the nucleus where it interacts with transcription factors and DNA-binding proteins that modulate target gene transcription. YAP1 TEAD Translocation Nucleus Interaction Transcription Factors DNA-binding Proteins Target Gene Transcription Complex Formation
1185	The US health care system can save up to $750 million if 7% of patients waiting for kidney transplants participate in the optimized national kidney paired donation program. The US health care system can save up to $750 million if 7% of patients waiting for kidney transplants participate in the optimized national kidney paired donation program. The US health care system can save up to $750 million if 7% of patients waiting for kidney transplants participate in the optimized national kidney paired donation program. The US health care system can save up to $750 million if 7% of patients waiting for kidney transplants participate in the optimized national kidney paired donation program. The US health care system can save up to $750 million if 7% of patients waiting for kidney transplants participate in the optimized national kidney paired donation program. Certainly! Here is a list of keywords for the given query US Health Care System $750 million Savings Kidney Transplants Patients Waiting List 7% National Kidney Paired Donation Program Optimized Transplantation Medical Costs Kidney Donors Health Policy Organ Donation Efficiency
1062	S-nitrosylated GAPDH physiologically transnitrosylates histone deacetylases. S-nitrosylated GAPDH physiologically transnitrosylates histone deacetylases. S-nitrosylated GAPDH physiologically transnitrosylates histone deacetylases. S-nitrosylated GAPDH physiologically transnitrosylates histone deacetylases. S-nitrosylated GAPDH physiologically transnitrosylates histone deacetylases. S-nitrosylation GAPDH Histone deacetylases Transnitrosylation Physiological Protein modification Nitric oxide Enzyme activity Post-translational modification Cell signaling Gene regulation Protein-protein interaction Epigenetics Enzymatic mechanism Redox biology
1180	The PRR MDA5 is a sensor of RNA virus infection. The PRR MDA5 is a sensor of RNA virus infection. The PRR MDA5 is a sensor of RNA virus infection. The PRR MDA5 is a sensor of RNA virus infection. The PRR MDA5 is a sensor of RNA virus infection. PRR MDA5 Sensor RNA Virus Infection Immune Response Pathogen Recognition Receptor Viral Detection Innate Immunity
198	CCL19 is absent within dLNs. CCL19 is absent within dLNs. CCL19 is absent within dLNs. CCL19 is absent within dLNs. CCL19 is absent within dLNs. CCL19 Absent dLNs Lymph Nodes Chemokine Expression Immunology Tissue Distribution Molecular Biology Lymph Node Microenvironment
870	Obesity decreases life quality. Obesity decreases life quality. Obesity decreases life quality. Obesity decreases life quality. Obesity decreases life quality. Obesity Life quality Health impact Well-being Physical health Mental health Lifestyle Chronic diseases Mobility Social interactions Self-esteem Productivity Longevity Medical complications Daily activities
993	Pyridostatin destabilizes the G - quadruplex in the telomeric region. Pyridostatin destabilizes the G - quadruplex in the telomeric region. Pyridostatin destabilizes the G - quadruplex in the telomeric region. Pyridostatin destabilizes the G - quadruplex in the telomeric region. Pyridostatin destabilizes the G - quadruplex in the telomeric region. Pyridostatin Destabilizes G-quadruplex Telomeric Region Telomeres Quadruplex DNA Molecular Biology Biochemistry
873	Obesity is determined solely by environmental factors. Obesity is determined solely by environmental factors. Obesity is determined solely by environmental factors. Obesity is determined solely by environmental factors. Obesity is determined solely by environmental factors. Obesity Environmental factors Determinants Solely Influence Lifestyle Diet Physical activity Genetics (for contrast) Non-genetic factors
1179	"The PRR MDA5 has a central DExD/H RNA helices domain. The PRR MDA5 has a central DExD/H RNA helices domain. The PRR MDA5 has a central DExD/H RNA helices domain. The PRR MDA5 has a central DExD/H RNA helices domain. The PRR MDA5 has a central DExD/H RNA helices domain. Certainly! Here is a list of keywords for the query: ""The PRR MDA5 has a central DExD/H RNA helices domain PRR MDA5 Central DExD/H RNA Helices Domain"
1298	Thigh-length graduated compression stockings (GCS) did not reduce deep vein thrombosis in patients admitted to hospital who are immobile because of acute stroke. Thigh-length graduated compression stockings (GCS) did not reduce deep vein thrombosis in patients admitted to hospital who are immobile because of acute stroke. Thigh-length graduated compression stockings (GCS) did not reduce deep vein thrombosis in patients admitted to hospital who are immobile because of acute stroke. Thigh-length graduated compression stockings (GCS) did not reduce deep vein thrombosis in patients admitted to hospital who are immobile because of acute stroke. Thigh-length graduated compression stockings (GCS) did not reduce deep vein thrombosis in patients admitted to hospital who are immobile because of acute stroke. Thigh-length Graduated compression stockings (GCS) Deep vein thrombosis (DVT) Hospital patients Immobility Acute stroke Prevention Thromboprophylaxis Clinical trial Efficacy Stroke recovery Venous thromboembolism (VTE) Medical compression Inpatient care Stroke rehabilitation
513	High cardiopulmonary fitness causes increased mortality rate. High cardiopulmonary fitness causes increased mortality rate. High cardiopulmonary fitness causes increased mortality rate. High cardiopulmonary fitness causes increased mortality rate. High cardiopulmonary fitness causes increased mortality rate. The query seems to present a counterintuitive idea as high cardiopulmonary fitness is generally associated with lower mortality rates. However if you are looking for keywords to explore this topic here's a list **Cardiopulmonary Fitness** **High Fitness Level** **Aerobic Capacity** **VO2 Max** **Mortality Rate** **Increased Mortality** **Physical Activity** **Exercise Intensity** **Cardiovascular Health** **Longevity** **Health Outcomes**
514	High dietary calcium intakes are unnecessary for prevention of secondary hyperparathyroidism in subjects with 25(OH)D levels above 75 nmol/liter. High dietary calcium intakes are unnecessary for prevention of secondary hyperparathyroidism in subjects with 25(OH)D levels above 75 nmol/liter. High dietary calcium intakes are unnecessary for prevention of secondary hyperparathyroidism in subjects with 25(OH)D levels above 75 nmol/liter. High dietary calcium intakes are unnecessary for prevention of secondary hyperparathyroidism in subjects with 25(OH)D levels above 75 nmol/liter. High dietary calcium intakes are unnecessary for prevention of secondary hyperparathyroidism in subjects with 25(OH)D levels above 75 nmol/liter. Certainly! Here is a list of keywords for the given query High dietary calcium Secondary hyperparathyroidism Prevention 25(OH)D levels 75 nmol/liter Unnecessary Subjects Vitamin D Calcium intake Hyperparathyroidism prevention Nutritional requirements Bone health Endocrine disorders Clinical nutrition Public health recommendations
756	Many proteins in human cells can be post-translationally modified at lysine residues via acetylation. Many proteins in human cells can be post-translationally modified at lysine residues via acetylation. Many proteins in human cells can be post-translationally modified at lysine residues via acetylation. Many proteins in human cells can be post-translationally modified at lysine residues via acetylation. Many proteins in human cells can be post-translationally modified at lysine residues via acetylation. Proteins Human cells Post-translational modification Lysine residues Acetylation
636	Inositol lipid 3-phosphatase PTEN converts Ptdlns(3,4)P 2 into phosphatidylinositol 4-phosphate. Inositol lipid 3-phosphatase PTEN converts Ptdlns(3,4)P 2 into phosphatidylinositol 4-phosphate. Inositol lipid 3-phosphatase PTEN converts Ptdlns(3,4)P 2 into phosphatidylinositol 4-phosphate. Inositol lipid 3-phosphatase PTEN converts Ptdlns(3,4)P 2 into phosphatidylinositol 4-phosphate. Inositol lipid 3-phosphatase PTEN converts Ptdlns(3,4)P 2 into phosphatidylinositol 4-phosphate. Inositol Lipid 3-phosphatase PTEN PtdIns(3 4)P2 Phosphatidylinositol 4-phosphate Conversion Enzyme Phosphatase
516	High levels of CRP reduces the risk of exacerbations in chronic obstructive pulmonary disease (COPD). High levels of CRP reduces the risk of exacerbations in chronic obstructive pulmonary disease (COPD). High levels of CRP reduces the risk of exacerbations in chronic obstructive pulmonary disease (COPD). High levels of CRP reduces the risk of exacerbations in chronic obstructive pulmonary disease (COPD). High levels of CRP reduces the risk of exacerbations in chronic obstructive pulmonary disease (COPD). CRP Chronic Obstructive Pulmonary Disease COPD Inflammation Exacerbations Risk Reduction Biomarkers C-Reactive Protein Respiratory Disease Clinical Outcomes
637	Input from  mental and physical health care professionals is effective at decreasing homelessness. Input from  mental and physical health care professionals is effective at decreasing homelessness. Input from  mental and physical health care professionals is effective at decreasing homelessness. Input from  mental and physical health care professionals is effective at decreasing homelessness. Input from  mental and physical health care professionals is effective at decreasing homelessness. Mental health professionals Physical health professionals Homelessness Decreasing homelessness Input from healthcare professionals Effectiveness Intervention Health care Mental health care Physical health care Homeless population Health interventions Professional input Community health Social services Housing support Recovery Treatment Preventive care Integrated care
879	Occupancy of ribosomes by IncRNAs do not make functional peptides. Occupancy of ribosomes by IncRNAs do not make functional peptides. Occupancy of ribosomes by IncRNAs do not make functional peptides. Occupancy of ribosomes by IncRNAs do not make functional peptides. Occupancy of ribosomes by IncRNAs do not make functional peptides. Ribosomes IncRNAs Occupancy Functional Peptides Non-coding RNAs Translation Gene Regulation Ribosome Binding Protein Synthesis RNA Interference Molecular Biology Transcription Post-transcriptional Regulation Long Non-coding RNAs (lncRNAs) Ribosome Profiling Cellular Processes RNA-Binding Proteins Gene Expression Bioinformatics
517	High levels of copeptin decrease risk of diabetes. High levels of copeptin decrease risk of diabetes. High levels of copeptin decrease risk of diabetes. High levels of copeptin decrease risk of diabetes. High levels of copeptin decrease risk of diabetes. Copeptin Diabetes Risk Reduction High Levels Biomarker Endocrine Insulin Resistance Cardiovascular Metabolic Syndrome Clinical Studies Glycemic Control Prognostic Indicator Hormonal Regulation Preventive Medicine Health Outcomes
759	Mathematical models predict that using Artemisinin-based combination therapy over nongametocytocidal drugs have a dramatic impact in reducing malaria transmission. Mathematical models predict that using Artemisinin-based combination therapy over nongametocytocidal drugs have a dramatic impact in reducing malaria transmission. Mathematical models predict that using Artemisinin-based combination therapy over nongametocytocidal drugs have a dramatic impact in reducing malaria transmission. Mathematical models predict that using Artemisinin-based combination therapy over nongametocytocidal drugs have a dramatic impact in reducing malaria transmission. Mathematical models predict that using Artemisinin-based combination therapy over nongametocytocidal drugs have a dramatic impact in reducing malaria transmission. Mathematical models Artemisinin-based combination therapy Nongametocytocidal drugs Malaria transmission Impact Reduction Treatment efficacy Disease modeling Public health Parasite transmission Gametocytes Antimalarial drugs Clinical outcomes Epidemiology Vector control
94	Albendazole is used to treat lymphatic filariasis. Albendazole is used to treat lymphatic filariasis. Albendazole is used to treat lymphatic filariasis. Albendazole is used to treat lymphatic filariasis. Albendazole is used to treat lymphatic filariasis. Albendazole Treatment Lymphatic Filariasis Medication Parasitic Infection Elephantiasis Nematode Antihelminthic Disease Management Tropical Medicine
99	Alizarin forms hydrogen bonds with residues involved in PGAM1 substrate binding. Alizarin forms hydrogen bonds with residues involved in PGAM1 substrate binding. Alizarin forms hydrogen bonds with residues involved in PGAM1 substrate binding. Alizarin forms hydrogen bonds with residues involved in PGAM1 substrate binding. Alizarin forms hydrogen bonds with residues involved in PGAM1 substrate binding. Alizarin Hydrogen bonds Residues PGAM1 Substrate binding Enzyme Molecular interaction Biochemistry
1197	The availability of safe places to study is not effective at decreasing homelessness. The availability of safe places to study is not effective at decreasing homelessness. The availability of safe places to study is not effective at decreasing homelessness. The availability of safe places to study is not effective at decreasing homelessness. The availability of safe places to study is not effective at decreasing homelessness. Safe places Study areas Homelessness Effectiveness Reduction Shelters Public spaces Academic environments Housing stability Social services Urban planning Community resources Policy impact Support systems Displacement
1196	The availability of safe places to study is effective at decreasing homelessness. The availability of safe places to study is effective at decreasing homelessness. The availability of safe places to study is effective at decreasing homelessness. The availability of safe places to study is effective at decreasing homelessness. The availability of safe places to study is effective at decreasing homelessness. Safe places Study spaces Homelessness Decrease Effectiveness Shelter Community centers Libraries Support services Accessibility Public policy Social impact Intervention Youth homelessness Urban planning
1194	The arm density of TatAd complexes is due to structural rearrangements within Class1 TatAd complexes such as the 'charge zipper mechanism'. The arm density of TatAd complexes is due to structural rearrangements within Class1 TatAd complexes such as the 'charge zipper mechanism'. The arm density of TatAd complexes is due to structural rearrangements within Class1 TatAd complexes such as the 'charge zipper mechanism'. The arm density of TatAd complexes is due to structural rearrangements within Class1 TatAd complexes such as the 'charge zipper mechanism'. The arm density of TatAd complexes is due to structural rearrangements within Class1 TatAd complexes such as the 'charge zipper mechanism'. Arm density TatAd complexes Structural rearrangements Class1 Charge zipper mechanism
1191	The amount of publicly available DNA data doubles every 10 years. The amount of publicly available DNA data doubles every 10 years. The amount of publicly available DNA data doubles every 10 years. The amount of publicly available DNA data doubles every 10 years. The amount of publicly available DNA data doubles every 10 years. DNA Data Publicly Available Doubling Every 10 Years Growth Genetics Genomics Biological Information Exponential Increase
880	"Occupancy of ribosomes by IncRNAs mirror 5 0-UTRs Occupancy of ribosomes by IncRNAs mirror 5 0-UTRs Occupancy of ribosomes by IncRNAs mirror 5 0-UTRs Occupancy of ribosomes by IncRNAs mirror 5 0-UTRs Occupancy of ribosomes by IncRNAs mirror 5 0-UTRs Sure here is a list of keywords for the query ""Occupancy of ribosomes by IncRNAs mirror 5' UTRs"" Ribosomes IncRNAs Occupancy 5' UTRs Long Non-Coding RNAs (lncRNAs) Translation Regulation Gene Expression RNA-Binding Proteins Molecular Biology Transcriptomics Non-Coding RNAs RNA Structure RNA Function Ribosome Profiling"
882	Omnivores produce less trimethylamine N-oxide from dietary I-carnitine than vegetarians. Omnivores produce less trimethylamine N-oxide from dietary I-carnitine than vegetarians. Omnivores produce less trimethylamine N-oxide from dietary I-carnitine than vegetarians. Omnivores produce less trimethylamine N-oxide from dietary I-carnitine than vegetarians. Omnivores produce less trimethylamine N-oxide from dietary I-carnitine than vegetarians. Omnivores Vegetarians Trimethylamine N-oxide Dietary I-carnitine Production Metabolism Nutrition Diet Comparative study Biochemistry
641	Insomnia can be effectively treated with cognitive behavioral therapy. Insomnia can be effectively treated with cognitive behavioral therapy. Insomnia can be effectively treated with cognitive behavioral therapy. Insomnia can be effectively treated with cognitive behavioral therapy. Insomnia can be effectively treated with cognitive behavioral therapy. Insomnia Treatment Cognitive Behavioral Therapy Effectiveness Sleep Disorders Psychological Interventions Behavioral Techniques Therapy Cognitive Strategies Sleep Improvement
521	High-sensitivity cardiac troponin T (HSCT-T) dosage may not be diagnostic if the onset of symptoms occurs less than 3 hours before acute myocardial injury (AMI). High-sensitivity cardiac troponin T (HSCT-T) dosage may not be diagnostic if the onset of symptoms occurs less than 3 hours before acute myocardial injury (AMI). High-sensitivity cardiac troponin T (HSCT-T) dosage may not be diagnostic if the onset of symptoms occurs less than 3 hours before acute myocardial injury (AMI). High-sensitivity cardiac troponin T (HSCT-T) dosage may not be diagnostic if the onset of symptoms occurs less than 3 hours before acute myocardial injury (AMI). High-sensitivity cardiac troponin T (HSCT-T) dosage may not be diagnostic if the onset of symptoms occurs less than 3 hours before acute myocardial injury (AMI). High-sensitivity Cardiac Troponin HSCT-T Dosage Diagnostic Onset Symptoms Less Than Hours Acute Myocardial Injury AMI
644	Insulin increases risk of severe kidney failure. Insulin increases risk of severe kidney failure. Insulin increases risk of severe kidney failure. Insulin increases risk of severe kidney failure. Insulin increases risk of severe kidney failure. Insulin Kidney failure Severe Risk Diabetes Complications Renal function Treatment Medication Health Studies Evidence Outcomes Clinical trials Long-term effects
887	Only a minority of cells survive development after differentiation into stress-resistant spores. Only a minority of cells survive development after differentiation into stress-resistant spores. Only a minority of cells survive development after differentiation into stress-resistant spores. Only a minority of cells survive development after differentiation into stress-resistant spores. Only a minority of cells survive development after differentiation into stress-resistant spores. Minority Cells Survive Development Differentiation Stress-resistant Spores
525	Histone demethylase recruitment and a transient decrease in histone methylation is necessary for ligand-dependent induction of transcription by nuclear receptors. Histone demethylase recruitment and a transient decrease in histone methylation is necessary for ligand-dependent induction of transcription by nuclear receptors. Histone demethylase recruitment and a transient decrease in histone methylation is necessary for ligand-dependent induction of transcription by nuclear receptors. Histone demethylase recruitment and a transient decrease in histone methylation is necessary for ligand-dependent induction of transcription by nuclear receptors. Histone demethylase recruitment and a transient decrease in histone methylation is necessary for ligand-dependent induction of transcription by nuclear receptors. Certainly! Here's a list of keywords for the given query Histone demethylase Recruitment Transient decrease Histone methylation Ligand-dependent induction Transcription Nuclear receptors Gene regulation Chromatin modification Epigenetics Molecular biology Cell signaling Hormone response Transcriptional activation Protein-protein interactions Biological mechanisms Cellular processes Nuclear receptor signaling
768	Mercaptopurine is anabolized into the inactive methylmercaptopurine by thiopurine methyltrasnferase (TPMT). Mercaptopurine is anabolized into the inactive methylmercaptopurine by thiopurine methyltrasnferase (TPMT). Mercaptopurine is anabolized into the inactive methylmercaptopurine by thiopurine methyltrasnferase (TPMT). Mercaptopurine is anabolized into the inactive methylmercaptopurine by thiopurine methyltrasnferase (TPMT). Mercaptopurine is anabolized into the inactive methylmercaptopurine by thiopurine methyltrasnferase (TPMT). Certainly! Here is a list of keywords for the query Mercaptopurine Anabolized Inactive Methylmercaptopurine Thiopurine Methyltransferase TPMT Enzyme Metabolism Drug Metabolism
527	Homozygous deletion of murine Sbds gene from osterix-expressing mesenchymal stem and progenitor cells (MPCs) prevents oxidative stress. Homozygous deletion of murine Sbds gene from osterix-expressing mesenchymal stem and progenitor cells (MPCs) prevents oxidative stress. Homozygous deletion of murine Sbds gene from osterix-expressing mesenchymal stem and progenitor cells (MPCs) prevents oxidative stress. Homozygous deletion of murine Sbds gene from osterix-expressing mesenchymal stem and progenitor cells (MPCs) prevents oxidative stress. Homozygous deletion of murine Sbds gene from osterix-expressing mesenchymal stem and progenitor cells (MPCs) prevents oxidative stress. Certainly! Here is a list of keywords for the query Homozygous deletion Murine Sbds gene Osterix Mesenchymal stem cells Progenitor cells (MPCs) Oxidative stress Gene knockout Bone development Stem cell biology Genetic modification Cellular stress Developmental biology Molecular genetics Mouse model
528	Human T-lymphotropic virus type-I-associated myelopathy / tropical spastic paraparesis (HAM/TSP) patients produce Immunoglobulin G (IgG) antibodies which cross-react with an immunodominant epitope in Tax. Human T-lymphotropic virus type-I-associated myelopathy / tropical spastic paraparesis (HAM/TSP) patients produce Immunoglobulin G (IgG) antibodies which cross-react with an immunodominant epitope in Tax. Human T-lymphotropic virus type-I-associated myelopathy / tropical spastic paraparesis (HAM/TSP) patients produce Immunoglobulin G (IgG) antibodies which cross-react with an immunodominant epitope in Tax. Human T-lymphotropic virus type-I-associated myelopathy / tropical spastic paraparesis (HAM/TSP) patients produce Immunoglobulin G (IgG) antibodies which cross-react with an immunodominant epitope in Tax. Human T-lymphotropic virus type-I-associated myelopathy / tropical spastic paraparesis (HAM/TSP) patients produce Immunoglobulin G (IgG) antibodies which cross-react with an immunodominant epitope in Tax. Human T-lymphotropic virus type-I HTLV-I HAM/TSP Tropical spastic paraparesis Myelopathy Immunoglobulin G IgG Antibodies Cross-react Immunodominant epitope Tax protein HTLV-1-associated myelopathy Neurological disorder Viral infection Immune response
649	Integrating classroom-based collaborative learning with Web-based collaborative learning leads to subpar class performance Integrating classroom-based collaborative learning with Web-based collaborative learning leads to subpar class performance Integrating classroom-based collaborative learning with Web-based collaborative learning leads to subpar class performance Integrating classroom-based collaborative learning with Web-based collaborative learning leads to subpar class performance Integrating classroom-based collaborative learning with Web-based collaborative learning leads to subpar class performance Certainly! Here is a list of keywords for the given query Classroom-based Collaborative learning Web-based Integration Subpar performance Class performance Educational technology Blended learning Student engagement Learning outcomes Digital collaboration Traditional classroom Online learning Hybrid learning Academic achievement
1088	Silencing of Bcl2 is important for the maintenance and progression of tumors. Silencing of Bcl2 is important for the maintenance and progression of tumors. Silencing of Bcl2 is important for the maintenance and progression of tumors. Silencing of Bcl2 is important for the maintenance and progression of tumors. Silencing of Bcl2 is important for the maintenance and progression of tumors. Silencing Bcl2 Tumor maintenance Tumor progression Gene regulation Cancer Oncogenesis Apoptosis Cell death Therapy resistance Molecular biology Genetic inhibition Tumor biology
1086	Sildenafil improves erectile function in men who experience sexual dysfunction as a result of the use of SSRI antidepressants. Sildenafil improves erectile function in men who experience sexual dysfunction as a result of the use of SSRI antidepressants. Sildenafil improves erectile function in men who experience sexual dysfunction as a result of the use of SSRI antidepressants. Sildenafil improves erectile function in men who experience sexual dysfunction as a result of the use of SSRI antidepressants. Sildenafil improves erectile function in men who experience sexual dysfunction as a result of the use of SSRI antidepressants. Sildenafil Erectile function Men Sexual dysfunction SSRI antidepressants Improvement Treatment Sexual health Antidepressants Erectile dysfunction
770	Metastatic colorectal cancer treated with a single agent fluoropyrimidines resulted in reduced efficacy and lower quality of life when compared with oxaliplatin-based chemotherapy in elderly patients. Metastatic colorectal cancer treated with a single agent fluoropyrimidines resulted in reduced efficacy and lower quality of life when compared with oxaliplatin-based chemotherapy in elderly patients. Metastatic colorectal cancer treated with a single agent fluoropyrimidines resulted in reduced efficacy and lower quality of life when compared with oxaliplatin-based chemotherapy in elderly patients. Metastatic colorectal cancer treated with a single agent fluoropyrimidines resulted in reduced efficacy and lower quality of life when compared with oxaliplatin-based chemotherapy in elderly patients. Metastatic colorectal cancer treated with a single agent fluoropyrimidines resulted in reduced efficacy and lower quality of life when compared with oxaliplatin-based chemotherapy in elderly patients. Metastatic colorectal cancer Single agent fluoropyrimidines Reduced efficacy Lower quality of life Oxaliplatin-based chemotherapy Elderly patients Treatment comparison Chemotherapy outcomes Fluoropyrimidines Oxaliplatin Cancer therapy Elderly oncology Clinical outcomes Treatment efficacy Quality of life in cancer patients
410	Febrile seizures increase the threshold for development of epilepsy. Febrile seizures increase the threshold for development of epilepsy. Febrile seizures increase the threshold for development of epilepsy. Febrile seizures increase the threshold for development of epilepsy. Febrile seizures increase the threshold for development of epilepsy. Febrile seizures Epilepsy Threshold Development Risk factors Neurological disorders Seizure predisposition Brain injury Long-term outcomes Pediatric neurology
411	"Febrile seizures reduce the threshold for development of epilepsy. Febrile seizures reduce the threshold for development of epilepsy. Febrile seizures reduce the threshold for development of epilepsy. Febrile seizures reduce the threshold for development of epilepsy. Febrile seizures reduce the threshold for development of epilepsy. Sure here is a list of keywords related to the query ""Febrile seizures reduce the threshold for development of epilepsy"" Febrile seizures Epilepsy Threshold Development Neurological disorders Seizures Risk factors Childhood epilepsy Brain development Neural damage Long-term effects Neural mechanisms Medical research Clinical studies Prognosis Prevention Treatment Recurrence"
532	Hyperfibrinogenemia decreases rates of femoropopliteal bypass thrombosis. Hyperfibrinogenemia decreases rates of femoropopliteal bypass thrombosis. Hyperfibrinogenemia decreases rates of femoropopliteal bypass thrombosis. Hyperfibrinogenemia decreases rates of femoropopliteal bypass thrombosis. Hyperfibrinogenemia decreases rates of femoropopliteal bypass thrombosis. Certainly! Here is a list of keywords for the given query Hyperfibrinogenemia Femoropopliteal Bypass Thrombosis Rates Decreases Vascular surgery Blood clotting Fibrinogen levels Peripheral artery disease (PAD) Surgical outcomes Hemostasis Cardiovascular risk Prognosis Clinical studies
533	Hyperfibrinogenemia increases rates of femoropopliteal bypass thrombosis. Hyperfibrinogenemia increases rates of femoropopliteal bypass thrombosis. Hyperfibrinogenemia increases rates of femoropopliteal bypass thrombosis. Hyperfibrinogenemia increases rates of femoropopliteal bypass thrombosis. Hyperfibrinogenemia increases rates of femoropopliteal bypass thrombosis. Hyperfibrinogenemia Femoropopliteal Bypass Thrombosis Rates Vascular Surgery Blood Clotting Cardiovascular Diseases Peripheral Artery Disease Hemostasis Fibrinogen Levels Surgical Complications Thromboembolic Events Prothrombotic States Clinical Outcomes
775	Mice defective for deoxyribonucleic acid (DNA) polymerase I (polI) reveal increased sensitivity to ionizing radiation (IR). Mice defective for deoxyribonucleic acid (DNA) polymerase I (polI) reveal increased sensitivity to ionizing radiation (IR). Mice defective for deoxyribonucleic acid (DNA) polymerase I (polI) reveal increased sensitivity to ionizing radiation (IR). Mice defective for deoxyribonucleic acid (DNA) polymerase I (polI) reveal increased sensitivity to ionizing radiation (IR). Mice defective for deoxyribonucleic acid (DNA) polymerase I (polI) reveal increased sensitivity to ionizing radiation (IR). Certainly! Here is a list of keywords for the query Mice Defective Deoxyribonucleic acid DNA Polymerase I polI Increased sensitivity Ionizing radiation IR Genetic mutation DNA repair Radiosensitivity Cellular response Mutagenesis Genomic stability These keywords can help in searching for relevant literature or articles related to the query
1199	The benefits of colchicine were achieved with effective widespread use of secondary prevention strategies such as high-dose statins. The benefits of colchicine were achieved with effective widespread use of secondary prevention strategies such as high-dose statins. The benefits of colchicine were achieved with effective widespread use of secondary prevention strategies such as high-dose statins. The benefits of colchicine were achieved with effective widespread use of secondary prevention strategies such as high-dose statins. The benefits of colchicine were achieved with effective widespread use of secondary prevention strategies such as high-dose statins. Colchicine Benefits Secondary prevention Strategies High-dose statins Widespread use Cardiovascular disease Prevention Drug therapy Clinical outcomes Medical treatment Risk reduction Cardiovascular events Anti-inflammatory Statin therapy
535	Hypertension is frequently observed in type 1 diabetes patients. Hypertension is frequently observed in type 1 diabetes patients. Hypertension is frequently observed in type 1 diabetes patients. Hypertension is frequently observed in type 1 diabetes patients. Hypertension is frequently observed in type 1 diabetes patients. Hypertension Type 1 Diabetes Frequency Observation Patients Comorbidity Cardiovascular Risk Blood Pressure Insulin-dependent Diabetes Mellitus (IDDM) Clinical Manifestations
415	Female carriers of the Apolipoprotein E4 (APOE4) allele have increased risk for dementia. Female carriers of the Apolipoprotein E4 (APOE4) allele have increased risk for dementia. Female carriers of the Apolipoprotein E4 (APOE4) allele have increased risk for dementia. Female carriers of the Apolipoprotein E4 (APOE4) allele have increased risk for dementia. Female carriers of the Apolipoprotein E4 (APOE4) allele have increased risk for dementia. Female Carriers Apolipoprotein E4 APOE4 Allele Increased risk Dementia Genetic Neurodegenerative Cognitive decline
536	"Hypocretin neurones induce panicprone state in rats. Hypocretin neurones induce panicprone state in rats. Hypocretin neurones induce panicprone state in rats. Hypocretin neurones induce panicprone state in rats. Hypocretin neurones induce panicprone state in rats. Certainly! Here is a list of keywords for the query ""Hypocretin neurones induce panic-prone state in rats"" Hypocretin Neurones Panic-prone Rats Induce State Anxiety Neuroscience Behavioral Neurotransmitters Experimental Stress Brain Physiology Research Animal Model Hypothalamus Neural Circuits"
659	"Ivermectin is used to treat lymphatic filariasis. Ivermectin is used to treat lymphatic filariasis. Ivermectin is used to treat lymphatic filariasis. Ivermectin is used to treat lymphatic filariasis. Ivermectin is used to treat lymphatic filariasis. Certainly! Here is a list of keywords for the query ""Ivermectin is used to treat lymphatic filariasis"" Ivermectin Treatment Lymphatic filariasis Parasitic disease Medication Antiparasitic Filariasis Drug therapy PublicInset sector Tropical disease These keywords can help in searching for more information or conducting research on the topic"
539	Hypoglycemia increases the risk of dementia. Hypoglycemia increases the risk of dementia. Hypoglycemia increases the risk of dementia. Hypoglycemia increases the risk of dementia. Hypoglycemia increases the risk of dementia. Hypoglycemia Dementia Risk Cognitive decline Brain function Insulin Blood sugar Neurodegeneration Elderly Diabetes complications Cognitive impairment Memory loss Brain health Glucose levels Metabolic disorders Neurological disorders Long-term effects Prevention Treatment Research findings
1099	Statins decrease blood cholesterol. Statins decrease blood cholesterol. Statins decrease blood cholesterol. Statins decrease blood cholesterol. Statins decrease blood cholesterol. Statins Blood Cholesterol Decrease Lipid-lowering Cardiovascular Pharmaceuticals Treatment Health Medication
660	Ivermectin is used to treat onchocerciasis. Ivermectin is used to treat onchocerciasis. Ivermectin is used to treat onchocerciasis. Ivermectin is used to treat onchocerciasis. Ivermectin is used to treat onchocerciasis. Ivermectin Onchocerciasis Treatment Medication Parasitic infection River blindness Antiparasitic Tropical disease Drug therapy Filariasis
781	Mice that lack Interferon-γ or its receptor exhibit high resistance to experimental autoimmune myocarditis. Mice that lack Interferon-γ or its receptor exhibit high resistance to experimental autoimmune myocarditis. Mice that lack Interferon-γ or its receptor exhibit high resistance to experimental autoimmune myocarditis. Mice that lack Interferon-γ or its receptor exhibit high resistance to experimental autoimmune myocarditis. Mice that lack Interferon-γ or its receptor exhibit high resistance to experimental autoimmune myocarditis. Mice Interferon-γ Receptor Lack High resistance Experimental autoimmune myocarditis Immune response Autoimmune disease Myocarditis Genetic knockout Inflammation Cardiac pathology Cytokine deficiency Animal model Immune modulation
540	"Hypothalamic glutamate neurotransmission is crucial to energy balance. Hypothalamic glutamate neurotransmission is crucial to energy balance. Hypothalamic glutamate neurotransmission is crucial to energy balance. Hypothalamic glutamate neurotransmission is crucial to energy balance. Hypothalamic glutamate neurotransmission is crucial to energy balance. Certainly! Here is a list of keywords for the query ""Hypothalamic glutamate neurotransmission is crucial to energy balance"" Hypothalamus Glutamate Neurotransmission Energy Balance Metabolism Appetite Regulation Feeding Behavior Obesity Neurons Synaptic Transmission Neuropeptides Homeostasis Central Nervous System (CNS) Brain Function Glutamate Receptors"
783	"Mice without IFN-γ or its receptor are resistant to EAM induced with α-MyHC/CFA. Mice without IFN-γ or its receptor are resistant to EAM induced with α-MyHC/CFA. Mice without IFN-γ or its receptor are resistant to EAM induced with α-MyHC/CFA. Mice without IFN-γ or its receptor are resistant to EAM induced with α-MyHC/CFA. Mice without IFN-γ or its receptor are resistant to EAM induced with α-MyHC/CFA. Certainly! Here is a list of keywords for the query ""Mice without IFN-γ or its receptor are resistant to EAM induced with α-MyHC/CFA"" Mice IFN-γ (Interferon-gamma) IFN-γ receptor EAM (Experimental Autoimmune Myocarditis) α-MyHC (Alpha-Myosin Heavy Chain) CFA (Complete Freund's Adjuvant) Resistance Autoimmune Myocarditis Immunology Genetic knockout"
300	Cytosolic proteins bind to iron-responsive elements on mRNAs coding for DMT1. Cytosolic proteins bind to iron-responsive elements on mRNAs coding for proteins involved in iron uptake. Cytosolic proteins bind to iron-responsive elements on mRNAs coding for DMT1. Cytosolic proteins bind to iron-responsive elements on mRNAs coding for proteins involved in iron uptake. Cytosolic proteins bind to iron-responsive elements on mRNAs coding for DMT1. Cytosolic proteins bind to iron-responsive elements on mRNAs coding for proteins involved in iron uptake. Cytosolic proteins bind to iron-responsive elements on mRNAs coding for DMT1. Cytosolic proteins bind to iron-responsive elements on mRNAs coding for proteins involved in iron uptake. Cytosolic proteins bind to iron-responsive elements on mRNAs coding for DMT1. Cytosolic proteins bind to iron-responsive elements on mRNAs coding for proteins involved in iron uptake. Sure here is a list of keywords for the given query Cytosolic proteins Iron-responsive elements mRNAs DMT1 Iron uptake Proteins Binding Gene regulation Iron metabolism mRNA stability Translational control Iron homeostasis Ferritin Transferrin receptor Iron transport Cellular iron Post-transcriptional regulation RNA-binding proteins
421	Flexible molecules experience greater steric hindrance in the tumor microenviroment than rigid molecules. Flexible molecules experience greater steric hindrance in the tumor microenviroment than rigid molecules. Flexible molecules experience greater steric hindrance in the tumor microenviroment than rigid molecules. Flexible molecules experience greater steric hindrance in the tumor microenviroment than rigid molecules. Flexible molecules experience greater steric hindrance in the tumor microenviroment than rigid molecules. Certainly! Here is a list of keywords that are relevant to the given query Flexible molecules Rigid molecules Steric hindrance Tumor microenvironment Molecular flexibility Molecular rigidity Tumor penetration Drug delivery Cancer therapy Molecular dynamics Bioavailability Physicochemical properties Conformational changes Molecular interactions Tumor cell environment These keywords should help in focusing research or discussions around the topic of how molecular flexibility affects st
784	"MicroRNA is involved in the regulation of Neural Stem Cell (NSC) differentiation and proliferation dynamic homeostasis MicroRNA is involved in the regulation of Neural Stem Cell (NSC) differentiation and proliferation dynamic homeostasis MicroRNA is involved in the regulation of Neural Stem Cell (NSC) differentiation and proliferation dynamic homeostasis MicroRNA is involved in the regulation of Neural Stem Cell (NSC) differentiation and proliferation dynamic homeostasis MicroRNA is involved in the regulation of Neural Stem Cell (NSC) differentiation and proliferation dynamic homeostasis Certainly! Here is a list of keywords for the query ""MicroRNA is involved in the regulation of Neural Stem Cell (NSC) differentiation and proliferation dynamic homeostasis"" MicroRNA miRNA Neural Stem Cells NSC Differentiation Proliferation Regulation Dynamic Homeostasis Stem Cell Biology Neurogenesis miRNA Targets miRNA Pathways Cell Cycle Gene Expression Developmental Biology"
785	Microarray results from culture-amplified mixtures of serotypes correlate poorly with microarray results from uncultured mixtures. Microarray results from culture-amplified mixtures of serotypes correlate poorly with microarray results from uncultured mixtures. Microarray results from culture-amplified mixtures of serotypes correlate poorly with microarray results from uncultured mixtures. Microarray results from culture-amplified mixtures of serotypes correlate poorly with microarray results from uncultured mixtures. Microarray results from culture-amplified mixtures of serotypes correlate poorly with microarray results from uncultured mixtures. Microarray Culture-amplified Mixtures Serotypes Correlate Poorly Uncultured Results Comparison Diagnostic Molecular Serotyping Bacterial Samples Amplification Hybridization Genetic Variability Sensitivity Specificity
544	IFIT1 restricts viral replication by sequestrating mis-capped viral RNAs. IFIT1 restricts viral replication by sequestrating mis-capped viral RNAs. IFIT1 restricts viral replication by sequestrating mis-capped viral RNAs. IFIT1 restricts viral replication by sequestrating mis-capped viral RNAs. IFIT1 restricts viral replication by sequestrating mis-capped viral RNAs. IFIT1 Viral replication Restricts Sequestrating Mis-capped Viral RNAs Antiviral RNA binding Immune response Gene expression Viral infection Host defense Transcript modification Interferon-induced Post-transcriptional regulation
303	DMRT1 is a sex-determining gene that is epigenetically regulated by the MHM region. DMRT1 is a sex-determining gene that is epigenetically regulated by the MHM region. DMRT1 is a sex-determining gene that is epigenetically regulated by the MHM region. DMRT1 is a sex-determining gene that is epigenetically regulated by the MHM region. DMRT1 is a sex-determining gene that is epigenetically regulated by the MHM region. Certainly! Here is a list of keywords for the query DMRT1 Sex-determining gene Epigenetic regulation MHM region Gene expression Chromatin structure DNA methylation Histone modification Sexual development Genetic regulation Epigenetics Genomic imprinting Molecular biology Developmental biology Genetics
1089	Smc5/6 engagment drives the activation of SUMO E3 ligase Mms21 by ATP-dependent remolding. Smc5/6 engagment drives the activation of SUMO E3 ligase Mms21 by ATP-dependent remolding. Smc5/6 engagment drives the activation of SUMO E3 ligase Mms21 by ATP-dependent remolding. Smc5/6 engagment drives the activation of SUMO E3 ligase Mms21 by ATP-dependent remolding. Smc5/6 engagment drives the activation of SUMO E3 ligase Mms21 by ATP-dependent remolding. SMC5/6 Engagement Activation SUMO E3 Ligase Mms21 ATP-dependent Remodeling Molecular Biology Protein Interaction Enzyme Activation ATP Chromosome Stability Post-translational Modification SUMOylation
549	"IRG1 has antiviral effects against neurotropic viruses. IRG1 has antiviral effects against neurotropic viruses. IRG1 has antiviral effects against neurotropic viruses. IRG1 has antiviral effects against neurotropic viruses. IRG1 has antiviral effects against neurotropic viruses. Certainly! Here is a list of keywords related to the query ""IRG1 has antiviral effects against neurotropic viruses"" IRG1 Antiviral Neurotropic Viruses Immune Response GTPase 1 Viral Infection Neurological Viruses Antiviral Activity Pathogenesis Host Defense Viral Replication Neuroinvasion Cytokines Interferons Innate"
551	ITAM phosphorylation prevents the transfer of the T cell receptor (TCR) signal from the echo-domain to the cytoplasmic tail of the T cell receptor (TCR). ITAM phosphorylation prevents the transfer of the T cell receptor (TCR) signal from the echo-domain to the cytoplasmic tail of the T cell receptor (TCR). ITAM phosphorylation prevents the transfer of the T cell receptor (TCR) signal from the echo-domain to the cytoplasmic tail of the T cell receptor (TCR). ITAM phosphorylation prevents the transfer of the T cell receptor (TCR) signal from the echo-domain to the cytoplasmic tail of the T cell receptor (TCR). ITAM phosphorylation prevents the transfer of the T cell receptor (TCR) signal from the echo-domain to the cytoplasmic tail of the T cell receptor (TCR). Certainly! Here is a list of keywords for the given query ITAM Phosphorylation T cell receptor (TCR) Signal transfer Echo-domain Cytoplasmic tail Immunoreceptor Signaling pathway T-cell activation Immunology
793	Mitochondria are uninvolved in apoptosis. Mitochondria are uninvolved in apoptosis. Mitochondria are uninvolved in apoptosis. Mitochondria are uninvolved in apoptosis. Mitochondria are uninvolved in apoptosis. Mitochondria Uninvolved Apoptosis Cellular Death Organelle Function Biochemistry Cell Biology Signaling Pathways Research Disproven Misconception
431	"FoxO3a activation in neuronal death is mediated by reactive oxygen species (ROS). FoxO3a activation in neuronal death is mediated by reactive oxygen species (ROS). FoxO3a activation in neuronal death is mediated by reactive oxygen species (ROS). FoxO3a activation in neuronal death is mediated by reactive oxygen species (ROS). FoxO3a activation in neuronal death is mediated by reactive oxygen species (ROS). Certainly! Here is a list of keywords for the query: ""FoxO3a activation in neuronal death is mediated by reactive oxygen species (ROS) FoxO3a Activation Neuronal Death Reactive Oxygen Species (ROS) Mediation Neurodegeneration Oxidative Stress Cell Signaling Neuroprotection Apoptosis Neurological Disorders Antioxidants Transcription Factors Brain Cells Molecular Mechanisms"
552	IgA plasma cells that are specific for transglutaminase 2 accumulate in the duodenal mucosa on commencement of a gluten-free diet. IgA plasma cells that are specific for transglutaminase 2 accumulate in the duodenal mucosa on commencement of a gluten-free diet. IgA plasma cells that are specific for transglutaminase 2 accumulate in the duodenal mucosa on commencement of a gluten-free diet. IgA plasma cells that are specific for transglutaminase 2 accumulate in the duodenal mucosa on commencement of a gluten-free diet. IgA plasma cells that are specific for transglutaminase 2 accumulate in the duodenal mucosa on commencement of a gluten-free diet. IgA Plasma cells Transglutaminase 2 Duodenal mucosa Gluten-free diet Accumulation Celiac disease (implied context)
674	LDL cholesterol has no involvement in the development of cardiovascular disease. LDL cholesterol has no involvement in the development of cardiovascular disease. LDL cholesterol has no involvement in the development of cardiovascular disease. LDL cholesterol has no involvement in the development of cardiovascular disease. LDL cholesterol has no involvement in the development of cardiovascular disease. LDL Cholesterol Cardiovascular Disease Involvement Development Atherosclerosis Lipoprotein Heart Health Blood Vessels Risk Factors Misconception
312	"De novo assembly of sequence data has more specific contigs than unassembled sequence data. De novo assembly of sequence data has more specific contigs than unassembled sequence data. De novo assembly of sequence data has more specific contigs than unassembled sequence data. De novo assembly of sequence data has more specific contigs than unassembled sequence data. De novo assembly of sequence data has more specific contigs than unassembled sequence data. Certainly! Here's a list of keywords for the query: ""De novo assembly of sequence data has more specific contigs than unassembled sequence data De novo assembly Sequence data Contigs Unassembled sequence Genomics Bioinformatics DNA sequencing Assembly algorithms Genome assembly Sequence alignment NGS (Next-Generation Sequencing) Read mapping Contig length Scaffold Sequence quality Assembly accuracy"
554	Immune complex triggered cell death leads to extracellular release of neutrophil protein HMGB1. Immune complex triggered cell death leads to extracellular release of neutrophil protein HMGB1. Immune complex triggered cell death leads to extracellular release of neutrophil protein HMGB1. Immune complex triggered cell death leads to extracellular release of neutrophil protein HMGB1. Immune complex triggered cell death leads to extracellular release of neutrophil protein HMGB1. Certainly! Here is a list of keywords relevant to the query Immune complex Cell death Extracellular release Neutrophil Protein HMGB1 Inflammation Immune response Neutrophil extracellular traps (NETs) Cytokine release Autoimmunity Apoptosis Necroptosis Inflammatory signaling Immune-mediated diseases Tissue damage
314	Deamination of cytidine to uridine on the minus strand of viral DNA results in catastrophic G-to-A mutations in the viral genome. Deamination of cytidine to uridine on the minus strand of viral DNA results in catastrophic G-to-A mutations in the viral genome. Deamination of cytidine to uridine on the minus strand of viral DNA results in catastrophic G-to-A mutations in the viral genome. Deamination of cytidine to uridine on the minus strand of viral DNA results in catastrophic G-to-A mutations in the viral genome. Deamination of cytidine to uridine on the minus strand of viral DNA results in catastrophic G-to-A mutations in the viral genome. Deamination Cytidine Uridine Minus strand Viral DNA G-to-A mutations Viral genome Catastrophic mutations
436	Free histones are degraded by a Rad53-dependent mechanism once DNA has been replicated. Free histones are degraded by a Rad53-dependent mechanism once DNA has been replicated. Free histones are degraded by a Rad53-dependent mechanism once DNA has been replicated. Free histones are degraded by a Rad53-dependent mechanism once DNA has been replicated. Free histones are degraded by a Rad53-dependent mechanism once DNA has been replicated. Free histones Degradation Rad53 Mechanism DNA replication Cell cycle Chromatin assembly Protein degradation Checkpoint kinase Replication-coupled histone turnover
437	Functional consequences of genomic alterations due to Myelodysplastic syndrome (MDS) are poorly understood due to the lack of an animal model. Functional consequences of genomic alterations due to Myelodysplastic syndrome (MDS) are poorly understood due to the lack of an animal model. Functional consequences of genomic alterations due to Myelodysplastic syndrome (MDS) are poorly understood due to the lack of an animal model. Functional consequences of genomic alterations due to Myelodysplastic syndrome (MDS) are poorly understood due to the lack of an animal model. Functional consequences of genomic alterations due to Myelodysplastic syndrome (MDS) are poorly understood due to the lack of an animal model. Myelodysplastic syndrome Genomic alterations Functional consequences Animal model Poorly understood MDS Genetic mutations Disease mechanisms Preclinical models Hematopoietic stem cells Chromosomal abnormalities Gene expression Molecular pathways Cancer biology Translational research
439	Fz/PCP-dependent Pk localizes to the anterior membrane of neuroectoderm cells during zebrafish neuralation Fz/PCP-dependent Pk localizes to the anterior membrane of neuroectoderm cells during zebrafish neuralation Fz/PCP-dependent Pk localizes to the anterior membrane of neuroectoderm cells during zebrafish neuralation Fz/PCP-dependent Pk localizes to the anterior membrane of neuroectoderm cells during zebrafish neuralation Fz/PCP-dependent Pk localizes to the anterior membrane of neuroectoderm cells during zebrafish neuralation Fz PCP Pk Anterior membrane Neuroectoderm cells Zebrafish Neuralation Localization Development Cell polarity Morphogenesis Tissue patterning Embryonic development Planar cell polarity pathway Neural tube formation
560	Immune responses result in the development of inflammatory Th17 cells and anti-inflammatory iTregs. Immune responses result in the development of inflammatory Th17 cells and anti-inflammatory iTregs. Immune responses result in the development of inflammatory Th17 cells and anti-inflammatory iTregs. Immune responses result in the development of inflammatory Th17 cells and anti-inflammatory iTregs. Immune responses result in the development of inflammatory Th17 cells and anti-inflammatory iTregs. Certainly! Here is a list of keywords for the given query Immune responses Inflammatory Th17 cells Anti-inflammatory iTregs Development Immune cells T-cell differentiation Cytokines Immune regulation Inflammation Regulatory T cells Adaptive immunity Immune modulation Cell-mediated immunity
440	Fz/PCP-dependent Pk localizes to the anterior membrane of notochord cells during zebrafish neuralation. Fz/PCP-dependent Pk localizes to the anterior membrane of notochord cells during zebrafish neuralation. Fz/PCP-dependent Pk localizes to the anterior membrane of notochord cells during zebrafish neuralation. Fz/PCP-dependent Pk localizes to the anterior membrane of notochord cells during zebrafish neuralation. Fz/PCP-dependent Pk localizes to the anterior membrane of notochord cells during zebrafish neuralation. Certainly! Here is a list of keywords for the given query Fz PCP (Planar Cell Polarity) Pk (Prickle) Anterior membrane Notochord cells Zebrafish Neurulation Localization Developmental biology Embryogenesis These keywords should help in identifying and organizing relevant information for the specified query
1303	Tirasemtiv has no effect on fast-twitch muscle. Tirasemtiv has no effect on fast-twitch muscle. Tirasemtiv has no effect on fast-twitch muscle. Tirasemtiv has no effect on fast-twitch muscle. Tirasemtiv has no effect on fast-twitch muscle. Tirasemtiv Fast-twitch Muscle Effect No effect Pharmacology Muscle fibers Skeletal muscle Drug response Muscle contraction Clinical trials Motor function Muscle strength Treatment Myopathy
684	"Lack of clpC does not affect sporulation efficiency in Bacillus subtilis cells. Lack of clpC does not affect sporulation efficiency in Bacillus subtilis cells. Lack of clpC does not affect sporulation efficiency in Bacillus subtilis cells. Lack of clpC does not affect sporulation efficiency in Bacillus subtilis cells. Lack of clpC does not affect sporulation efficiency in Bacillus subtilis cells. Certainly! Here is a list of keywords for the query ""Lack of clpC does not affect sporulation efficiency in Bacillus subtilis cells"" ClpC Sporulation Efficiency Bacillus subtilis Gene deletion Mutant Bacterial genetics Molecular biology Microbiology Developmental biology Protein function Stress response Cell cycle Spore formation Genetic regulation"
443	"GATA-3 is important for hematopoietic stem cell (HSC) function. GATA-3 is important for hematopoietic stem cell (HSC) function. GATA-3 is important for hematopoietic stem cell (HSC) function. GATA-3 is important for hematopoietic stem cell (HSC) function. GATA-3 is important for hematopoietic stem cell (HSC) function. Certainly! Here is a list of keywords for the query ""GATA-3 is important for hematopoietic stem cell (HSC) function"" GATA-3 Hematopoietic stem cells HSC Function Importance Transcription factor Blood cell development Hematopoiesis Stem cell maintenance Differentiation Regulation Gene expression Cell fate Lineage commitment Immune system Bone"
324	Deleting Raptor reduces G-CSF levels. Deleting Raptor reduces G-CSF levels. Deleting Raptor reduces G-CSF levels. Deleting Raptor reduces G-CSF levels. Deleting Raptor reduces G-CSF levels. Raptor Deletion G-CSF Levels Reduction Knockout Gene Cytokine Granulocyte-colony stimulating factor Molecular signaling Pathway Biological response Inhibition Experimental model Therapeutic target
327	Deletion of αvβ8 does not result in a spontaneous inflammatory phenotype. Deletion of αvβ8 does not result in a spontaneous inflammatory phenotype. Deletion of αvβ8 does not result in a spontaneous inflammatory phenotype. Deletion of αvβ8 does not result in a spontaneous inflammatory phenotype. Deletion of αvβ8 does not result in a spontaneous inflammatory phenotype. Deletion αvβ8 Spontaneous Inflammatory Phenotype Absence Inflammation Integrin Gene Knockout Mice Immune Response Pathology Tissue Homeostasis Disease Model Experimental Biology
569	In adult tissue, most T cells are memory T cells. In adult tissue, most T cells are memory T cells. In adult tissue, most T cells are memory T cells. In adult tissue, most T cells are memory T cells. In adult tissue, most T cells are memory T cells. Adult tissue T cells Memory T cells Immune system Lymphocytes Adaptive immunity Immunological memory Cell-mediated immunity T lymphocytes Peripheral blood Lymphoid organs T-cell development T-cell activation T-cell subsets Longevity of T cells
208	"CHEK2 is not associated with breast cancer. CHEK2 is not associated with breast cancer. CHEK2 is not associated with breast cancer. CHEK2 is not associated with breast cancer. CHEK2 is not associated with breast cancer. Certainly! Here is a list of keywords for the query ""CHEK2 is not associated with breast cancer"" CHEK2 Breast cancer Non-association Genetic predisposition Cancer genetics Mutation Oncology Risk factors Genomic studies Clinical research Genetic testing Hereditary cancer Tumor suppression DNA repair Biomarkers"
690	Less than 10% of the gabonese children with Schimmelpenning-Feuerstein-Mims syndrome (SFM) had a plasma lactate of more than 5mmol/L. Less than 10% of the gabonese children with Schimmelpenning-Feuerstein-Mims syndrome (SFM) had a plasma lactate of more than 5mmol/L. Less than 10% of the gabonese children with Schimmelpenning-Feuerstein-Mims syndrome (SFM) had a plasma lactate of more than 5mmol/L. Less than 10% of the gabonese children with Schimmelpenning-Feuerstein-Mims syndrome (SFM) had a plasma lactate of more than 5mmol/L. Less than 10% of the gabonese children with Schimmelpenning-Feuerstein-Mims syndrome (SFM) had a plasma lactate of more than 5mmol/L. Gabonese Children Schimmelpenning-Feuerstein-Mims syndrome SFM Plasma lactate 5mmol/L Less than 10%
691	Leukemia associated Rho guanine nucleotide-exchange factor represses RhoA in response to SRC activation. Leukemia associated Rho guanine nucleotide-exchange factor represses RhoA in response to SRC activation. Leukemia associated Rho guanine nucleotide-exchange factor represses RhoA in response to SRC activation. Leukemia associated Rho guanine nucleotide-exchange factor represses RhoA in response to SRC activation. Leukemia associated Rho guanine nucleotide-exchange factor represses RhoA in response to SRC activation. Leukemia Rho guanine nucleotide-exchange factor (GEF) RhoA SRC activation Repression Signaling pathway Protein interaction Oncogene Cellular regulation Cancer biology Molecular mechanism Guanine nucleotide exchange Rho GTPase Cell transformation Tumor progression
692	"Leuko-increased blood increases infectious complications in red blood cell transfusion. Leuko-increased blood increases infectious complications in red blood cell transfusion. Leuko-increased blood increases infectious complications in red blood cell transfusion. Leuko-increased blood increases infectious complications in red blood cell transfusion. Leuko-increased blood increases infectious complications in red blood cell transfusion. Certainly! Here are the keywords for the query ""Leuko-increased blood increases infectious complications in red blood cell transfusion"" Leuko Leukocytes Blood Infectious Complications Red blood cell Transfusion Increased White blood cells Immune response Transfusion-related Infection Hematology Medical Pathology"
1316	"Transferred UCB T cells acquire a memory-like phenotype in recipients. Transferred UCB T cells acquire a memory-like phenotype in recipients. Transferred UCB T cells acquire a memory-like phenotype in recipients. Transferred UCB T cells acquire a memory-like phenotype in recipients. Transferred UCB T cells acquire a memory-like phenotype in recipients. Certainly! Here is a list of keywords for the query: ""Transferred UCB T cells acquire a memory-like phenotype in recipients Transferred UCB T cells Umbilical Cord Blood T cells Memory-like phenotype Recipients Immunotherapy Cell transfer Phenotypic changes Memory T cells Adaptive immunity Immune response Cellular therapy Hematopoietic stem cells Clinical applications Immune"
693	Leuko-reduced blood reduces infectious complications in red blood cell transfusion. Leuko-reduced blood reduces infectious complications in red blood cell transfusion. Leuko-reduced blood reduces infectious complications in red blood cell transfusion. Leuko-reduced blood reduces infectious complications in red blood cell transfusion. Leuko-reduced blood reduces infectious complications in red blood cell transfusion. Leuko-reduction Blood transfusion Red blood cells Infectious complications Leukocytes Transfusion safety Hematological disorders Clinical outcomes Pathogen reduction Transfusion-associated infections
452	Gene expression does not vary appreciably across genetically identical cells. Gene expression does not vary appreciably across genetically identical cells. Gene expression does not vary appreciably across genetically identical cells. Gene expression does not vary appreciably across genetically identical cells. Gene expression does not vary appreciably across genetically identical cells. Gene expression Genetic identity Cellular homogeneity Transcriptome Identical cells Variability Consistency RNA levels Genetic uniformity Expression stability
212	"CR is associated with higher methylation age. CR is associated with higher methylation age. CR is associated with higher methylation age. CR is associated with higher methylation age. CR is associated with higher methylation age. Certainly! Here is a list of keywords for the query ""CR is associated with higher methylation age"" CR Caloric Restriction Methylation Age Epigenetic Age DNA Methylation Aging Biomarkers Epigenetics Lifespan Healthspan Cellular Aging Biological Age Molecular Aging Longevity Gene Expression Chromatin Modification Histone Modification Age-related Changes"
575	In domesticated populations of Saccharomyces cerevisiae, whole chromosome aneuploidy is very uncommon. In domesticated populations of Saccharomyces cerevisiae, whole chromosome aneuploidy is very uncommon. In domesticated populations of Saccharomyces cerevisiae, whole chromosome aneuploidy is very uncommon. In domesticated populations of Saccharomyces cerevisiae, whole chromosome aneuploidy is very uncommon. In domesticated populations of Saccharomyces cerevisiae, whole chromosome aneuploidy is very uncommon. Saccharomyces cerevisiae Domesticated populations Whole chromosome aneuploidy Uncommon Yeast genetics Chromosomal variation Genetic stability Aneuploidy frequency Population genetics Yeast strains
213	CRP is not predictive of postoperative mortality following Coronary Artery Bypass Graft (CABG) surgery. CRP is not predictive of postoperative mortality following Coronary Artery Bypass Graft (CABG) surgery. CRP is not predictive of postoperative mortality following Coronary Artery Bypass Graft (CABG) surgery. CRP is not predictive of postoperative mortality following Coronary Artery Bypass Graft (CABG) surgery. CRP is not predictive of postoperative mortality following Coronary Artery Bypass Graft (CABG) surgery. Certainly! Here is a list of keywords related to the query CRP C-Reactive Protein Postoperative Mortality Coronary Artery Bypass Graft CABG Surgery Predictive Value Clinical Outcomes Cardiac Surgery Inflammatory Markers Risk Factors Surgical Complications Patient Outcomes Hospitalization Recovery Prognostic Indicators Medical Research
577	In mice, P. chabaudi parasites are able to proliferate faster early in infection when inoculated at lower numbers than when inoculated at high numbers. In mice, P. chabaudi parasites are able to proliferate faster early in infection when inoculated at lower numbers than when inoculated at high numbers. In mice, P. chabaudi parasites are able to proliferate faster early in infection when inoculated at lower numbers than when inoculated at high numbers. In mice, P. chabaudi parasites are able to proliferate faster early in infection when inoculated at lower numbers than when inoculated at high numbers. In mice, P. chabaudi parasites are able to proliferate faster early in infection when inoculated at lower numbers than when inoculated at high numbers. Mice P. chabaudi Parasites Proliferation Infection Inoculation Low numbers High numbers Early infection Parasite growth dynamics
578	In mouse models, the loss of CSF1R facilitates MOZ-TIF2-induced leuekmogenesis. In mouse models, the loss of CSF1R facilitates MOZ-TIF2-induced leuekmogenesis. In mouse models, the loss of CSF1R facilitates MOZ-TIF2-induced leuekmogenesis. In mouse models, the loss of CSF1R facilitates MOZ-TIF2-induced leuekmogenesis. In mouse models, the loss of CSF1R facilitates MOZ-TIF2-induced leuekmogenesis. Certainly! Here is a list of keywords for the given query Mouse models CSF1R CSF1R loss MOZ-TIF2 Leukemogenesis Oncogenesis Hematopoietic system Transcription factors Signal transduction Cancer biology Genetic knockout Myeloid cells Colony-stimulating factor 1 receptor Fusion protein Acute myeloid leukemia (AML) Molecular mechanisms
216	"CX3CR1 on the Th2 cells impairs T cell survival CX3CR1 on the Th2 cells impairs T cell survival CX3CR1 on the Th2 cells impairs T cell survival CX3CR1 on the Th2 cells impairs T cell survival CX3CR1 on the Th2 cells impairs T cell survival Certainly! Here is a list of keywords for the query ""CX3CR1 on the Th2 cells impairs T cell survival"" CX3CR1 Th2 cells T cell survival Immune response Chemokine receptor Inflammation Lymphocytes Apoptosis Signaling pathways Cytokines Immune modulation Cell death T helper cells Immune system Receptor-ligand interaction"
217	"CX3CR1 on the Th2 cells promotes T cell survival CX3CR1 on the Th2 cells promotes T cell survival CX3CR1 on the Th2 cells promotes T cell survival CX3CR1 on the Th2 cells promotes T cell survival CX3CR1 on the Th2 cells promotes T cell survival Certainly! Here is a list of keywords for the query: ""CX3CR1 on the Th2 cells promotes T cell survival"" **CX3CR1** **Th2 cells** **T cell survival** **Chemokine receptor** **Immune response** **Lymphocytes** **Activation** **Proliferation** **Apoptosis** **Inflammation** **Cell signaling** **Immune regulation** **Cytokines** **Gene"
338	Dexamethasone decreases risk of postoperative bleeding. Dexamethasone decreases risk of postoperative bleeding. Dexamethasone decreases risk of postoperative bleeding. Dexamethasone decreases risk of postoperative bleeding. Dexamethasone decreases risk of postoperative bleeding. Dexamethasone Postoperative bleeding Risk reduction Surgical complications Hemostasis Corticosteroids Perioperative care Clinical outcomes Blood loss Patient safety
218	CX3CR1 on the Th2 cells promotes airway inflammation. CX3CR1 on the Th2 cells promotes airway inflammation. CX3CR1 on the Th2 cells promotes airway inflammation. CX3CR1 on the Th2 cells promotes airway inflammation. CX3CR1 on the Th2 cells promotes airway inflammation. CX3CR1 Th2 cells Airway inflammation Chemokine receptor Immune response Allergic asthma Cytokines Inflammation signaling Respiratory diseases Immune regulation Asthma pathology Inflammatory mediators Lung inflammation T-helper cells Chemotaxis
219	"CX3CR1 on the Th2 cells suppresses airway inflammation. CX3CR1 on the Th2 cells suppresses airway inflammation. CX3CR1 on the Th2 cells suppresses airway inflammation. CX3CR1 on the Th2 cells suppresses airway inflammation. CX3CR1 on the Th2 cells suppresses airway inflammation. Certainly! Here is a list of keywords for the query ""CX3CR1 on the Th2 cells suppresses airway inflammation"" CX3CR1 Th2 cells Airway inflammation Suppression Immunology Chemokine receptor T helper cells Asthma Inflammatory response Lung Immune regulation Cytokines Allergic inflammation Respiratory diseases Immune signaling"
1319	Transplanted human glial cells can differentiate within the host animal. Transplanted human glial cells can differentiate within the host animal. Transplanted human glial cells can differentiate within the host animal. Transplanted human glial cells can differentiate within the host animal. Transplanted human glial cells can differentiate within the host animal. Transplanted Human Glial cells Differentiate Host animal Differentiation Xenotransplantation Glia Neural tissue Cell transplantation
100	"All hematopoietic stem cells segregate their chromosomes randomly. All hematopoietic stem cells segregate their chromosomes randomly. All hematopoietic stem cells segregate their chromosomes randomly. All hematopoietic stem cells segregate their chromosomes randomly. All hematopoietic stem cells segregate their chromosomes randomly. Certainly! Here is a list of keywords for the query: ""All hematopoietic stem cells segregate their chromosomes randomly Hematopoietic stem cells Chromosome segregation Random Cell division Genetics Hematopoiesis Mitosis DNA distribution Cell biology Stem cell research"
1204	The combination of H3K4me3 and H3K79me2 is found in quiescent hair follicle stem cells. The combination of H3K4me3 and H3K79me2 is found in quiescent hair follicle stem cells. The combination of H3K4me3 and H3K79me2 is found in quiescent hair follicle stem cells. The combination of H3K4me3 and H3K79me2 is found in quiescent hair follicle stem cells. The combination of H3K4me3 and H3K79me2 is found in quiescent hair follicle stem cells. Certainly! Here is a list of keywords for the given query H3K4me3 H3K79me2 Quiescent Hair follicle stem cells Combination Epigenetic marks Histone modifications Stem cell biology Gene regulation Quiescence maintenance
343	Diabetic patients with acute coronary syndrome experience increased short-term and long-term risk for bleeding events. Diabetic patients with acute coronary syndrome experience increased short-term and long-term risk for bleeding events. Diabetic patients with acute coronary syndrome experience increased short-term and long-term risk for bleeding events. Diabetic patients with acute coronary syndrome experience increased short-term and long-term risk for bleeding events. Diabetic patients with acute coronary syndrome experience increased short-term and long-term risk for bleeding events. Diabetic patients Acute coronary syndrome Increased risk Short-term risk Long-term risk Bleeding events Complications Cardiovascular Hemorrhage Thrombosis Anticoagulation Antiplatelet therapy Clinical outcomes Hemostasis Treatment Risk factors Mortality Morbidity Patient management Prognosis
1202	The center of the granuloma in an immune cell induces a pro-inflammatory immune response. The center of the granuloma in an immune cell induces a pro-inflammatory immune response. The center of the granuloma in an immune cell induces a pro-inflammatory immune response. The center of the granuloma in an immune cell induces a pro-inflammatory immune response. The center of the granuloma in an immune cell induces a pro-inflammatory immune response. Granuloma Center Immune cell Pro-inflammatory Immune response Induces
587	In transgenic mice harboring green florescent protein under the control of the Sox2 promoter, less than ten percent of the cells with green florescent colocalize with cell proliferation markers. In transgenic mice harboring green florescent protein under the control of the Sox2 promoter, less than ten percent of the cells with green florescent colocalize with cell proliferation markers. In transgenic mice harboring green florescent protein under the control of the Sox2 promoter, less than ten percent of the cells with green florescent colocalize with cell proliferation markers. In transgenic mice harboring green florescent protein under the control of the Sox2 promoter, less than ten percent of the cells with green florescent colocalize with cell proliferation markers. In transgenic mice harboring green florescent protein under the control of the Sox2 promoter, less than ten percent of the cells with green florescent colocalize with cell proliferation markers. Transgenic mice Green fluorescent protein (GFP) Sox2 promoter Cell proliferation markers Colocalization Less than ten percent Fluorescence Promoter activity Cell population Expression pattern
1200	The binding orientation of the ML-SA1 activator at hTRPML2 is different from the binding orientation of the ML-SA1 activator at hTRPML1. The binding orientation of the ML-SA1 activator at hTRPML2 is different from the binding orientation of the ML-SA1 activator at hTRPML1. The binding orientation of the ML-SA1 activator at hTRPML2 is different from the binding orientation of the ML-SA1 activator at hTRPML1. The binding orientation of the ML-SA1 activator at hTRPML2 is different from the binding orientation of the ML-SA1 activator at hTRPML1. The binding orientation of the ML-SA1 activator at hTRPML2 is different from the binding orientation of the ML-SA1 activator at hTRPML1. ML-SA1 Activator Binding orientation hTRPML2 hTRPML1 TRPML2 TRPML1 Differences Molecular interaction
589	In young and middle-aged adults, current or remote uses of ADHD medications do not increase the risk of serious cardiovascular events. In young and middle-aged adults, current or remote uses of ADHD medications do not increase the risk of serious cardiovascular events. In young and middle-aged adults, current or remote uses of ADHD medications do not increase the risk of serious cardiovascular events. In young and middle-aged adults, current or remote uses of ADHD medications do not increase the risk of serious cardiovascular events. In young and middle-aged adults, current or remote uses of ADHD medications do not increase the risk of serious cardiovascular events. ADHD medications Young adults Middle-aged adults Current use Remote use Cardiovascular events Risk Serious cardiovascular events Safety Pharmacotherapy Cardiovascular health Stimulants Non-stimulants Long-term effects Adverse effects Epidemiology Clinical outcomes Health risks Medication safety Age groups
1320	Transplanted human glial progenitor cells are incapable of forming a neural network with host animals' neurons. Transplanted human glial progenitor cells are incapable of forming a neural network with host animals' neurons. Transplanted human glial progenitor cells are incapable of forming a neural network with host animals' neurons. Transplanted human glial progenitor cells are incapable of forming a neural network with host animals' neurons. Transplanted human glial progenitor cells are incapable of forming a neural network with host animals' neurons. Transplanted Human Glial Progenitor Cells Incapable Forming Neural Network Host Animals Neurons Integration Neurobiology Cell Interaction Brain Tissue Neural Plasticity
903	"PD-1 triggering on monocytes reduces IL-10 production by monocytes. PD-1 triggering on monocytes reduces IL-10 production by monocytes. PD-1 triggering on monocytes reduces IL-10 production by monocytes. PD-1 triggering on monocytes reduces IL-10 production by monocytes. PD-1 triggering on monocytes reduces IL-10 production by monocytes. Certainly! Here is a list of keywords for the query: ""PD-1 triggering on monocytes reduces IL-10 production by monocytes"" PD-1 Monocytes IL-10 Production Reduction Triggering Immune Response Cytokines Inflammation Signal Transduction Immunoregulation Cell Signaling Immune Cells Anti-inflammatory Programmed Death-1 Mon"
904	PDPN promotes efficient motility along stromal surfaces by activating the C-type lectin receptor to rearrange the actin cytoskeleton in dendritic cells. PDPN promotes efficient motility along stromal surfaces by activating the C-type lectin receptor to rearrange the actin cytoskeleton in dendritic cells. PDPN promotes efficient motility along stromal surfaces by activating the C-type lectin receptor to rearrange the actin cytoskeleton in dendritic cells. PDPN promotes efficient motility along stromal surfaces by activating the C-type lectin receptor to rearrange the actin cytoskeleton in dendritic cells. PDPN promotes efficient motility along stromal surfaces by activating the C-type lectin receptor to rearrange the actin cytoskeleton in dendritic cells. PDPN (Podoplanin) Efficient motility Stromal surfaces C-type lectin receptor Actin cytoskeleton Dendritic cells Activation Rearrangement Immune cells Cell migration Surface interactions Cytoskeletal dynamics Immune response Signal transduction Cell adhesion
1207	The composition of myosin-II isoform switches from the polarizable B isoform to the more homogenous A isoform during hematopoietic differentiation. The composition of myosin-II isoform switches from the polarizable B isoform to the more homogenous A isoform during hematopoietic differentiation. The composition of myosin-II isoform switches from the polarizable B isoform to the more homogenous A isoform during hematopoietic differentiation. The composition of myosin-II isoform switches from the polarizable B isoform to the more homogenous A isoform during hematopoietic differentiation. The composition of myosin-II isoform switches from the polarizable B isoform to the more homogenous A isoform during hematopoietic differentiation. Myosin-II Isoform B isoform A isoform Polarizable Homogenous Hematopoietic Differentiation Composition Switch